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1.
High-energy compounds promote physiological processing of Alzheimer's amyloid-ß precursor protein and boost cell survival in culture.
J Neurochem
; 123(4): 525-31, 2012 Nov.
Artículo
Inglés
| MEDLINE | ID: mdl-22906069
2.
Evidence supporting the role of calpain in the α-processing of amyloid-ß precursor protein.
Biochem Biophys Res Commun
; 420(3): 530-5, 2012 Apr 13.
Artículo
Inglés
| MEDLINE | ID: mdl-22480599
3.
Our "energy-Ca(2+) signaling deficits" hypothesis and its explanatory potential for key features of Alzheimer's disease.
Front Aging Neurosci
; 6: 329, 2014.
Artículo
Inglés
| MEDLINE | ID: mdl-25489296
4.
High-energy compounds mobilize intracellular Ca2+ and activate calpain in cultured cells: is calpain an energy-dependent protease?
Brain Res Bull
; 102: 9-14, 2014 Mar.
Artículo
Inglés
| MEDLINE | ID: mdl-24508187
5.
Elevated [Ca2+]i levels occur with decreased calpain activity in aged fibroblasts and their reversal by energy-rich compounds: new paradigm for Alzheimer's disease prevention.
J Alzheimers Dis
; 37(4): 835-48, 2013.
Artículo
Inglés
| MEDLINE | ID: mdl-24122005
6.
What to look for beyond "pathogenic" factors in senile dementia? A functional deficiency of Ca²âº signaling.
J Alzheimers Dis
; 27(4): 679-89, 2011.
Artículo
Inglés
| MEDLINE | ID: mdl-21876251
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