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Biochem Biophys Res Commun ; 508(1): 97-101, 2019 01 01.
Artículo en Inglés | MEDLINE | ID: mdl-30471854

RESUMEN

Nobiletin has protective effects on cardiovascular diseases, but the mechanism is not clear. In this study, we examined whether nobiletin affects the expression of miR-590/LPL and its relative effects on lipid accumulation and pro-inflammatory cytokine secretion in human THP-1 macrophages. RT-qPCR analysis showed that nobiletin increased the expression of miR-590. Western blot analysis showed that nobiletin-suppressed LPL expression was enhanced by miR-590 mimic and abrogated by miR-590 inhibitor. Oil Red O staining and high-performance liquid chromatography assays showed that nobiletin attenuated lipid accumulation in macrophages. Treatment with nobiletin and miR-590 mimic decreased cellular lipid accumulation, whereas treatment with miR-590 inhibitor increased cellular lipid accumulation. ELISA illustrated that nobiletin alleviated pro-inflammatory cytokine secretion in macrophages as measured by, which was reduced by miR-590 mimic and increased by miR-590 inhibitor. In conclusion, nobiletin may alleviate lipid accumulation and secretion of pro-inflammatory cytokines by enhancing the inhibitory effect of miR-590 on LPL expression, suggesting a promising strategy for potential drug development for atherosclerosis.


Asunto(s)
Flavonas/farmacología , Metabolismo de los Lípidos/efectos de los fármacos , Lipoproteína Lipasa/metabolismo , MicroARNs/genética , MicroARNs/metabolismo , Aterosclerosis/tratamiento farmacológico , Aterosclerosis/genética , Aterosclerosis/metabolismo , Cardiotónicos/farmacología , Citocinas/metabolismo , Regulación hacia Abajo/efectos de los fármacos , Desarrollo de Medicamentos , Humanos , Mediadores de Inflamación/metabolismo , Lipoproteína Lipasa/antagonistas & inhibidores , Macrófagos/efectos de los fármacos , Macrófagos/metabolismo , Células THP-1 , Regulación hacia Arriba/efectos de los fármacos
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