Detalhe da pesquisa
1.
In vivo phenotypic screening: clinical proof of concept for a drug repositioning approach.
Drug Discov Today Technol
; 23: 45-52, 2017 Mar.
Artigo
Inglês
| MEDLINE | ID: mdl-28647085
2.
Derivatives of human complement component C3 for therapeutic complement depletion: a novel class of therapeutic agents.
Adv Exp Med Biol
; 632: 293-307, 2008.
Artigo
Inglês
| MEDLINE | ID: mdl-19025130
3.
Association of increased cortical soluble abeta42 levels with diffuse plaques after severe brain injury in humans.
Arch Neurol
; 64(4): 541-4, 2007 Apr.
Artigo
Inglês
| MEDLINE | ID: mdl-17420316
4.
Comparative analysis of cortical gene expression in mouse models of Alzheimer's disease.
Neurobiol Aging
; 27(3): 377-86, 2006 Mar.
Artigo
Inglês
| MEDLINE | ID: mdl-15927307
5.
Activation of c-Jun N-terminal kinase and p38 in an Alzheimer's disease model is associated with amyloid deposition.
J Neurosci
; 22(9): 3376-85, 2002 May 01.
Artigo
Inglês
| MEDLINE | ID: mdl-11978814
6.
CEP-11004, an inhibitor of the SAPK/JNK pathway, reduces TNF-alpha release from lipopolysaccharide-treated cells and mice.
Eur J Pharmacol
; 515(1-3): 179-87, 2005 May 16.
Artigo
Inglês
| MEDLINE | ID: mdl-15904918
7.
Changes in expression of amyloid precursor protein and interleukin-1beta after experimental traumatic brain injury in rats.
J Neurotrauma
; 19(12): 1555-67, 2002 Dec.
Artigo
Inglês
| MEDLINE | ID: mdl-12542857
8.
Caspase inhibition therapy abolishes brain trauma-induced increases in Abeta peptide: implications for clinical outcome.
Exp Neurol
; 197(2): 437-50, 2006 Feb.
Artigo
Inglês
| MEDLINE | ID: mdl-16300758
9.
Alzheimer's pathology in human temporal cortex surgically excised after severe brain injury.
Exp Neurol
; 190(1): 192-203, 2004 Nov.
Artigo
Inglês
| MEDLINE | ID: mdl-15473992