Detalhe da pesquisa
1.
Reduced levels of MRE11 cause disease phenotypes distinct from ataxia telangiectasia-like disorder.
Hum Mol Genet
; 2024 Jun 18.
Artigo
Inglês
| MEDLINE | ID: mdl-38888340
2.
Correction: Complex Breakpoints and Template Switching Associated with Non-canonical Termination of Homologous Recombination in Mammalian Cells.
PLoS Genet
; 12(12): e1006509, 2016 Dec.
Artigo
Inglês
| MEDLINE | ID: mdl-27959912
3.
Complex Breakpoints and Template Switching Associated with Non-canonical Termination of Homologous Recombination in Mammalian Cells.
PLoS Genet
; 12(11): e1006410, 2016 Nov.
Artigo
Inglês
| MEDLINE | ID: mdl-27832076
4.
Elevated inflammatory responses and targeted therapeutic intervention in a preclinical mouse model of ataxia-telangiectasia lung disease.
Sci Rep
; 11(1): 4268, 2021 02 19.
Artigo
Inglês
| MEDLINE | ID: mdl-33608602
5.
Mechanisms of double-strand break repair in somatic mammalian cells.
Biochem J
; 423(2): 157-68, 2009 Sep 25.
Artigo
Inglês
| MEDLINE | ID: mdl-19772495
6.
Reversible mislocalization of a disease-associated MRE11 splice variant product.
Sci Rep
; 8(1): 10121, 2018 07 04.
Artigo
Inglês
| MEDLINE | ID: mdl-29973640
7.
Recruitment and activation of the ATM kinase in the absence of DNA-damage sensors.
Nat Struct Mol Biol
; 22(9): 736-43, 2015 Sep.
Artigo
Inglês
| MEDLINE | ID: mdl-26280532
8.
Impact of histone H4 lysine 20 methylation on 53BP1 responses to chromosomal double strand breaks.
PLoS One
; 7(11): e49211, 2012.
Artigo
Inglês
| MEDLINE | ID: mdl-23209566