Detalhe da pesquisa
1.
HES1 is a novel downstream modifier of the SHH-GLI3 Axis in the development of preaxial polydactyly.
PLoS Genet
; 17(12): e1009982, 2021 12.
Artigo
Inglês
| MEDLINE | ID: mdl-34928956
2.
Risk factors for intracellular fatty accumulation in rotator cuff muscle: a histologic analysis.
J Shoulder Elbow Surg
; 33(4): e215-e222, 2024 Apr.
Artigo
Inglês
| MEDLINE | ID: mdl-37757906
3.
Rac1 activation controls nuclear localization of beta-catenin during canonical Wnt signaling.
Cell
; 133(2): 340-53, 2008 Apr 18.
Artigo
Inglês
| MEDLINE | ID: mdl-18423204
4.
Dysregulation of STAT3 signaling is associated with endplate-oriented herniations of the intervertebral disc in Adgrg6 mutant mice.
PLoS Genet
; 15(10): e1008096, 2019 10.
Artigo
Inglês
| MEDLINE | ID: mdl-31652254
5.
Intracellular biosynthesis of lipids and cholesterol by Scap and Insig in mesenchymal cells regulates long bone growth and chondrocyte homeostasis.
Development
; 145(13)2018 07 09.
Artigo
Inglês
| MEDLINE | ID: mdl-29899135
6.
Characterization complex collagen fiber architecture in knee joint using high-resolution diffusion imaging.
Magn Reson Med
; 84(2): 908-919, 2020 08.
Artigo
Inglês
| MEDLINE | ID: mdl-31962373
7.
Diffusion tractography of the rat knee at microscopic resolution.
Magn Reson Med
; 81(6): 3775-3786, 2019 06.
Artigo
Inglês
| MEDLINE | ID: mdl-30671998
8.
Cell type-specific effects of Notch signaling activation on intervertebral discs: Implications for intervertebral disc degeneration.
J Cell Physiol
; 233(7): 5431-5440, 2018 07.
Artigo
Inglês
| MEDLINE | ID: mdl-30025440
9.
HES factors regulate specific aspects of chondrogenesis and chondrocyte hypertrophy during cartilage development.
J Cell Sci
; 129(11): 2145-55, 2016 06 01.
Artigo
Inglês
| MEDLINE | ID: mdl-27160681
10.
TAK1 regulates SOX9 expression in chondrocytes and is essential for postnatal development of the growth plate and articular cartilages.
J Cell Sci
; 126(Pt 24): 5704-13, 2013 Dec 15.
Artigo
Inglês
| MEDLINE | ID: mdl-24144697
11.
Cartilage-specific RBPjκ-dependent and -independent Notch signals regulate cartilage and bone development.
Development
; 139(6): 1198-212, 2012 Mar.
Artigo
Inglês
| MEDLINE | ID: mdl-22354840
12.
Multiple hereditary exostoses (MHE): elucidating the pathogenesis of a rare skeletal disorder through interdisciplinary research.
Connect Tissue Res
; 55(2): 80-8, 2014 Apr.
Artigo
Inglês
| MEDLINE | ID: mdl-24409815
13.
RBP-Jκ-dependent Notch signaling is required for murine articular cartilage and joint maintenance.
Arthritis Rheum
; 65(10): 2623-33, 2013 Oct.
Artigo
Inglês
| MEDLINE | ID: mdl-23839930
14.
Troponin T3 expression in skeletal and smooth muscle is required for growth and postnatal survival: characterization of Tnnt3(tm2a(KOMP)Wtsi) mice.
Genesis
; 51(9): 667-75, 2013 Sep.
Artigo
Inglês
| MEDLINE | ID: mdl-23775847
15.
BMP2, but not BMP4, is crucial for chondrocyte proliferation and maturation during endochondral bone development.
J Cell Sci
; 124(Pt 20): 3428-40, 2011 Oct 15.
Artigo
Inglês
| MEDLINE | ID: mdl-21984813
16.
RBPjkappa-dependent Notch signaling regulates mesenchymal progenitor cell proliferation and differentiation during skeletal development.
Development
; 137(9): 1461-71, 2010 May.
Artigo
Inglês
| MEDLINE | ID: mdl-20335360
17.
Engineering superficial zone features in tissue engineered cartilage.
Biotechnol Bioeng
; 110(5): 1476-86, 2013 May.
Artigo
Inglês
| MEDLINE | ID: mdl-23239161
18.
Association Between Supraspinatus Tendon Retraction, Histologic Myofiber Size, and Supraspinatus Muscle Atrophy on MRI.
Am J Sports Med
; 51(8): 1997-2004, 2023 07.
Artigo
Inglês
| MEDLINE | ID: mdl-37260272
19.
Aged G Protein-Coupled Receptor Kinase 3 (Grk3)-Deficient Mice Exhibit Enhanced Osteoclastogenesis and Develop Bone Lesions Analogous to Human Paget's Disease of Bone.
Cells
; 12(7)2023 03 23.
Artigo
Inglês
| MEDLINE | ID: mdl-37048054
20.
Radiation-induced bone loss in mice is ameliorated by inhibition of HIF-2α in skeletal progenitor cells.
Sci Transl Med
; 15(724): eabo5217, 2023 11 29.
Artigo
Inglês
| MEDLINE | ID: mdl-38019933