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Zhongguo Yi Xue Ke Xue Yuan Xue Bao ; 38(3): 253-9, 2016 06 10.
Artigo em Inglês | MEDLINE | ID: mdl-27469907

RESUMO

Objective To study the effect of endometrial stem cells (EnSCs) derived cytokine cocktail (EdCC) on myocardial ischemic reperfusion injury (I/R) in a mouse model. Methods EdCC was concentrated from the culture medium of EnSCs with Millipore ultra-filtration technology and was administrated to a myocardial I/R mouse models through tail vein injection. The infarct area was determined by TTC/Evans Blue staining. The apoptotic cells were counted by TUNEL assay and the protein level of cleaved caspase 3 was evaluated by Western blotting. Results The EdCC extraction efficiency was (222.4±29.3) Μg/10(6) cells in every 24 h,but the protein gradually degraded under-80 ℃ storage. As compared with I/R group,100 Μg fresh EdCC decreased infarct area (P=0.001),reduced apoptotic nuclei in the infarct border (P=0.019),and inhibited cleaved caspase 3 expression (P=0.002). Increasing EdCC dosage did not further reduce the infarct area. The myocardial protective effect of EdCC diminished after 90 days' storage under-80 ℃. Conclusion EdCC reduces myocardial I/R injury through protecting cardiomyocytes from apoptosis within 30 days storage under-80 ℃.


Assuntos
Citocinas/uso terapêutico , Endométrio/citologia , Traumatismo por Reperfusão Miocárdica/terapia , Células-Tronco/citologia , Animais , Apoptose , Caspase 3/metabolismo , Células Cultivadas , Modelos Animais de Doenças , Feminino , Camundongos , Miócitos Cardíacos/efeitos dos fármacos
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