Detalhe da pesquisa
1.
Chemistry-First Approach for Nomination of Personalized Treatment in Lung Cancer.
Cell
; 173(4): 864-878.e29, 2018 05 03.
Artigo
Inglês
| MEDLINE | ID: mdl-29681454
2.
A new anthropometric index to predict percent body fat in young adults.
Public Health Nutr
; 23(9): 1507-1514, 2020 06.
Artigo
Inglês
| MEDLINE | ID: mdl-32172710
3.
Thioridazine specifically sensitizes drug-resistant cancer cells through highly increase in apoptosis and P-gp inhibition.
Tumour Biol
; 35(10): 9831-8, 2014 Oct.
Artigo
Inglês
| MEDLINE | ID: mdl-24989930
4.
Co-treatment with the anti-malarial drugs mefloquine and primaquine highly sensitizes drug-resistant cancer cells by increasing P-gp inhibition.
Biochem Biophys Res Commun
; 441(3): 655-60, 2013 Nov 22.
Artigo
Inglês
| MEDLINE | ID: mdl-24284282
5.
Low amount of salinomycin greatly increases Akt activation, but reduces activated p70S6K levels.
Int J Mol Sci
; 14(9): 17304-18, 2013 Aug 22.
Artigo
Inglês
| MEDLINE | ID: mdl-23975168
6.
Salinomycin sensitizes antimitotic drugs-treated cancer cells by increasing apoptosis via the prevention of G2 arrest.
Biochem Biophys Res Commun
; 418(1): 98-103, 2012 Feb 03.
Artigo
Inglês
| MEDLINE | ID: mdl-22244892
7.
Salinomycin, a p-glycoprotein inhibitor, sensitizes radiation-treated cancer cells by increasing DNA damage and inducing G2 arrest.
Invest New Drugs
; 30(4): 1311-8, 2012 Aug.
Artigo
Inglês
| MEDLINE | ID: mdl-21573958
8.
SOCS5 and SOCS6 have similar expression patterns in normal and cancer tissues.
Tumour Biol
; 33(1): 215-21, 2012 Feb.
Artigo
Inglês
| MEDLINE | ID: mdl-22081311
9.
Lower salinomycin concentration increases apoptotic detachment in high-density cancer cells.
Int J Mol Sci
; 13(10): 13169-82, 2012 Oct 12.
Artigo
Inglês
| MEDLINE | ID: mdl-23202945
10.
Erratum to: Thioridazine specifically sensitizes drug-resistant cancer cells through highly increase in apoptosis and P-gp inhibition.
Tumour Biol
; 36(9): 7331, 2015 Sep.
Artigo
Inglês
| MEDLINE | ID: mdl-26271665
11.
Integrated pharmaco-proteogenomics defines two subgroups in isocitrate dehydrogenase wild-type glioblastoma with prognostic and therapeutic opportunities.
Nat Commun
; 11(1): 3288, 2020 07 03.
Artigo
Inglês
| MEDLINE | ID: mdl-32620753
12.
SP600125, an inhibitor of Jnk pathway, reduces viability of relatively resistant cancer cells to doxorubicin.
Biochem Biophys Res Commun
; 387(3): 450-5, 2009 Sep 25.
Artigo
Inglês
| MEDLINE | ID: mdl-19607816
13.
Co-treatment of LY294002 or MK-2206 with AZD5363 Attenuates AZD5363-induced Increase in the Level of Phosphorylated AKT.
Anticancer Res
; 36(11): 5849-5858, 2016 11.
Artigo
Inglês
| MEDLINE | ID: mdl-27793908
14.
Attenuation of Colchicine Toxicity in Drug-resistant Cancer Cells by Co-treatment with Anti-malarial Drugs.
Anticancer Res
; 36(11): 5859-5866, 2016 11.
Artigo
Inglês
| MEDLINE | ID: mdl-27793909
15.
Anti-malarial Drugs Primaquine and Chloroquine Have Different Sensitization Effects with Anti-mitotic Drugs in Resistant Cancer Cells.
Anticancer Res
; 36(4): 1641-8, 2016 Apr.
Artigo
Inglês
| MEDLINE | ID: mdl-27069141
16.
Correction: Meta-Analysis of Large-Scale Toxicogenomic Data Finds Neuronal Regeneration Related Protein and Cathepsin D to Be Novel Biomarkers of Drug-Induced Toxicity.
PLoS One
; 11(9): e0163403, 2016.
Artigo
Inglês
| MEDLINE | ID: mdl-27632408
17.
Co-treatment of Salinomycin Sensitizes AZD5363-treated Cancer Cells Through Increased Apoptosis.
Anticancer Res
; 35(9): 4741-7, 2015 Sep.
Artigo
Inglês
| MEDLINE | ID: mdl-26254364
18.
Meta-Analysis of Large-Scale Toxicogenomic Data Finds Neuronal Regeneration Related Protein and Cathepsin D to Be Novel Biomarkers of Drug-Induced Toxicity.
PLoS One
; 10(9): e0136698, 2015.
Artigo
Inglês
| MEDLINE | ID: mdl-26335687
19.
Sensitization of cancer cells through reduction of total Akt and downregulation of salinomycin-induced pAkt, pGSk3ß, pTSC2, and p4EBP1 by cotreatment with MK-2206.
Biomed Res Int
; 2014: 295760, 2014.
Artigo
Inglês
| MEDLINE | ID: mdl-25114899
20.
SP600125 overcomes antimitotic drug-resistance in cancer cells by increasing apoptosis with independence of P-gp inhibition.
Eur J Pharmacol
; 723: 141-7, 2014 Jan 15.
Artigo
Inglês
| MEDLINE | ID: mdl-24333214