Detalhe da pesquisa
1.
Prenatal, but not Postnatal, Curcumin Administration Rescues Neuromorphological and Cognitive Alterations in Ts65Dn Down Syndrome Mice.
J Nutr
; 150(9): 2478-2489, 2020 09 01.
Artigo
Inglês
| MEDLINE | ID: mdl-32729926
2.
Prenatal Administration of Oleic Acid or Linolenic Acid Reduces Neuromorphological and Cognitive Alterations in Ts65dn Down Syndrome Mice.
J Nutr
; 150(6): 1631-1643, 2020 06 01.
Artigo
Inglês
| MEDLINE | ID: mdl-32243527
3.
Cerebellar alterations in a model of Down syndrome: The role of the Dyrk1A gene.
Neurobiol Dis
; 110: 206-217, 2018 02.
Artigo
Inglês
| MEDLINE | ID: mdl-29221819
4.
Anti-IL17 treatment ameliorates Down syndrome phenotypes in mice.
Brain Behav Immun
; 73: 235-251, 2018 10.
Artigo
Inglês
| MEDLINE | ID: mdl-29758264
5.
Normalizing the gene dosage of Dyrk1A in a mouse model of Down syndrome rescues several Alzheimer's disease phenotypes.
Neurobiol Dis
; 106: 76-88, 2017 Oct.
Artigo
Inglês
| MEDLINE | ID: mdl-28647555
6.
Chronic Melatonin Administration Reduced Oxidative Damage and Cellular Senescence in the Hippocampus of a Mouse Model of Down Syndrome.
Neurochem Res
; 41(11): 2904-2913, 2016 Nov.
Artigo
Inglês
| MEDLINE | ID: mdl-27450081
7.
Decreasing the Expression of GABAA α5 Subunit-Containing Receptors Partially Improves Cognitive, Electrophysiological, and Morphological Hippocampal Defects in the Ts65Dn Model of Down Syndrome.
Mol Neurobiol
; 55(6): 4745-4762, 2018 Jun.
Artigo
Inglês
| MEDLINE | ID: mdl-28717969
8.
Pre- and post-natal melatonin administration partially regulates brain oxidative stress but does not improve cognitive or histological alterations in the Ts65Dn mouse model of Down syndrome.
Behav Brain Res
; 334: 142-154, 2017 09 15.
Artigo
Inglês
| MEDLINE | ID: mdl-28743603