Detalhe da pesquisa
1.
Sexually dimorphic effects of SARM1 deletion on cardiac NAD+ metabolism and function.
Am J Physiol Heart Circ Physiol
; 323(4): H774-H781, 2022 10 01.
Artigo
Inglês
| MEDLINE | ID: mdl-36053750
2.
Harnessing NAD+ Metabolism as Therapy for Cardiometabolic Diseases.
Curr Heart Fail Rep
; 19(4): 157-169, 2022 08.
Artigo
Inglês
| MEDLINE | ID: mdl-35556214
3.
Mitochondrial protein interactome elucidated by chemical cross-linking mass spectrometry.
Proc Natl Acad Sci U S A
; 114(7): 1732-1737, 2017 02 14.
Artigo
Inglês
| MEDLINE | ID: mdl-28130547
4.
Normalization of NAD+ Redox Balance as a Therapy for Heart Failure.
Circulation
; 134(12): 883-94, 2016 09 20.
Artigo
Inglês
| MEDLINE | ID: mdl-27489254
5.
Simultaneous Analysis of Major Coenzymes of Cellular Redox Reactions and Energy Using ex Vivo (1)H NMR Spectroscopy.
Anal Chem
; 88(9): 4817-24, 2016 05 03.
Artigo
Inglês
| MEDLINE | ID: mdl-27043450
6.
Revealing Pathway Dynamics in Heart Diseases by Analyzing Multiple Differential Networks.
PLoS Comput Biol
; 11(6): e1004332, 2015 Jun.
Artigo
Inglês
| MEDLINE | ID: mdl-26083688
7.
BIGH3 protein and macrophages in retinal endothelial cell apoptosis.
Apoptosis
; 20(1): 29-37, 2015 Jan.
Artigo
Inglês
| MEDLINE | ID: mdl-25378215
8.
Failed Power Plant Turns Into Mass Murder: New Insight on Mitochondrial Cardiomyopathy.
Circ Res
; 122(1): 11-13, 2018 01 05.
Artigo
Inglês
| MEDLINE | ID: mdl-29301837
9.
Mitochondrion as a Target for Heart Failure Therapy- Role of Protein Lysine Acetylation.
Circ J
; 79(9): 1863-70, 2015.
Artigo
Inglês
| MEDLINE | ID: mdl-26248514
10.
Redox regulation of MAPK phosphatase 1 controls monocyte migration and macrophage recruitment.
Proc Natl Acad Sci U S A
; 109(41): E2803-12, 2012 Oct 09.
Artigo
Inglês
| MEDLINE | ID: mdl-22991462
11.
Promoting PGC-1α-driven mitochondrial biogenesis is detrimental in pressure-overloaded mouse hearts.
Am J Physiol Heart Circ Physiol
; 307(9): H1307-16, 2014 Nov 01.
Artigo
Inglês
| MEDLINE | ID: mdl-25172896
12.
NADPH oxidase 4 mediates monocyte priming and accelerated chemotaxis induced by metabolic stress.
Arterioscler Thromb Vasc Biol
; 32(2): 415-26, 2012 Feb.
Artigo
Inglês
| MEDLINE | ID: mdl-22095986
13.
Late-life Rapamycin Treatment Enhances Cardiomyocyte Relaxation Kinetics and Reduces Myocardial Stiffness.
bioRxiv
; 2023 Jun 13.
Artigo
Inglês
| MEDLINE | ID: mdl-37398078
14.
Genetic architecture of heart mitochondrial proteome influencing cardiac hypertrophy.
Elife
; 122023 06 05.
Artigo
Inglês
| MEDLINE | ID: mdl-37276142
15.
Nox4 is a novel inducible source of reactive oxygen species in monocytes and macrophages and mediates oxidized low density lipoprotein-induced macrophage death.
Circ Res
; 106(9): 1489-97, 2010 May 14.
Artigo
Inglês
| MEDLINE | ID: mdl-20360249
16.
NAD+ Redox Imbalance in the Heart Exacerbates Diabetic Cardiomyopathy.
Circ Heart Fail
; 14(8): e008170, 2021 08.
Artigo
Inglês
| MEDLINE | ID: mdl-34374300
17.
Thiol oxidative stress induced by metabolic disorders amplifies macrophage chemotactic responses and accelerates atherogenesis and kidney injury in LDL receptor-deficient mice.
Arterioscler Thromb Vasc Biol
; 29(11): 1779-86, 2009 Nov.
Artigo
Inglês
| MEDLINE | ID: mdl-19592463
18.
Targeting NAD+ Metabolism as Interventions for Mitochondrial Disease.
Sci Rep
; 9(1): 3073, 2019 02 28.
Artigo
Inglês
| MEDLINE | ID: mdl-30816177
19.
Chemical Crosslinking Mass Spectrometry Analysis of Protein Conformations and Supercomplexes in Heart Tissue.
Cell Syst
; 6(1): 136-141.e5, 2018 Jan 24.
Artigo
Inglês
| MEDLINE | ID: mdl-29199018
20.
Electrostatic interactions contribute to reduced heat capacity change of unfolding in a thermophilic ribosomal protein l30e.
J Mol Biol
; 348(2): 419-31, 2005 Apr 29.
Artigo
Inglês
| MEDLINE | ID: mdl-15811378