Detalhe da pesquisa
1.
Axonal response of mitochondria to demyelination and complex IV activity within demyelinated axons in experimental models of multiple sclerosis.
Neuropathol Appl Neurobiol
; 49(1): e12851, 2023 Feb.
Artigo
Inglês
| MEDLINE | ID: mdl-36181265
2.
Enhanced axonal response of mitochondria to demyelination offers neuroprotection: implications for multiple sclerosis.
Acta Neuropathol
; 140(2): 143-167, 2020 08.
Artigo
Inglês
| MEDLINE | ID: mdl-32572598
3.
Cell type-specific Nrf2 expression in multiple sclerosis lesions.
Acta Neuropathol
; 130(2): 263-77, 2015 Aug.
Artigo
Inglês
| MEDLINE | ID: mdl-26087903
4.
Targeting the TCA cycle can ameliorate widespread axonal energy deficiency in neuroinflammatory lesions.
Nat Metab
; 5(8): 1364-1381, 2023 08.
Artigo
Inglês
| MEDLINE | ID: mdl-37430025
5.
Crucial neuroprotective roles of the metabolite BH4 in dopaminergic neurons.
bioRxiv
; 2023 May 08.
Artigo
Inglês
| MEDLINE | ID: mdl-37214873
6.
Phenotypic drug screen uncovers the metabolic GCH1/BH4 pathway as key regulator of EGFR/KRAS-mediated neuropathic pain and lung cancer.
Sci Transl Med
; 14(660): eabj1531, 2022 08 31.
Artigo
Inglês
| MEDLINE | ID: mdl-36044597
7.
Targeting mitochondria to protect axons in progressive MS.
Neurosci Lett
; 710: 134258, 2019 09 25.
Artigo
Inglês
| MEDLINE | ID: mdl-31082453