Detalhe da pesquisa
1.
Maresin 1 protects the liver against ischemia/reperfusion injury via the ALXR/Akt signaling pathway.
Mol Med
; 27(1): 18, 2021 02 25.
Artigo
Inglês
| MEDLINE | ID: mdl-33632134
2.
Immune-Responsive Gene 1/Itaconate Activates Nuclear Factor Erythroid 2-Related Factor 2 in Hepatocytes to Protect Against Liver Ischemia-Reperfusion Injury.
Hepatology
; 72(4): 1394-1411, 2020 10.
Artigo
Inglês
| MEDLINE | ID: mdl-31997373
3.
cGAS-mediated autophagy protects the liver from ischemia-reperfusion injury independently of STING.
Am J Physiol Gastrointest Liver Physiol
; 314(6): G655-G667, 2018 06 01.
Artigo
Inglês
| MEDLINE | ID: mdl-29446653
4.
Interferon ß (IFN-ß) Production during the Double-stranded RNA (dsRNA) Response in Hepatocytes Involves Coordinated and Feedforward Signaling through Toll-like Receptor 3 (TLR3), RNA-dependent Protein Kinase (PKR), Inducible Nitric Oxide Synthase (iNOS), and Src Protein.
J Biol Chem
; 291(29): 15093-107, 2016 07 15.
Artigo
Inglês
| MEDLINE | ID: mdl-27226571
5.
CaMKIV-dependent preservation of mTOR expression is required for autophagy during lipopolysaccharide-induced inflammation and acute kidney injury.
J Immunol
; 193(5): 2405-15, 2014 Sep 01.
Artigo
Inglês
| MEDLINE | ID: mdl-25070845
6.
A role for cGMP in inducible nitric-oxide synthase (iNOS)-induced tumor necrosis factor (TNF) α-converting enzyme (TACE/ADAM17) activation, translocation, and TNF receptor 1 (TNFR1) shedding in hepatocytes.
J Biol Chem
; 287(43): 35887-98, 2012 Oct 19.
Artigo
Inglês
| MEDLINE | ID: mdl-22898814
7.
PEX7 and EBP50 target iNOS to the peroxisome in hepatocytes.
Nitric Oxide
; 31: 9-19, 2013 May 31.
Artigo
Inglês
| MEDLINE | ID: mdl-23474170
8.
Characterization of DISC formation and TNFR1 translocation to mitochondria in TNF-α-treated hepatocytes.
Am J Pathol
; 179(3): 1221-9, 2011 Sep.
Artigo
Inglês
| MEDLINE | ID: mdl-21741934
9.
Heme oxygenase-1-mediated autophagy protects against hepatocyte cell death and hepatic injury from infection/sepsis in mice.
Hepatology
; 53(6): 2053-62, 2011 Jun.
Artigo
Inglês
| MEDLINE | ID: mdl-21437926
11.
A Tissue Perfusion Harvest Model for Optimal Multisystem Comparisons of Pathobiology.
Curr Protoc
; 2(1): e343, 2022 Jan.
Artigo
Inglês
| MEDLINE | ID: mdl-35030299
12.
Immunohistochemistry.
Curr Protoc
; 2(9): e549, 2022 Sep.
Artigo
Inglês
| MEDLINE | ID: mdl-36102926
13.
Reduced cleavage of von willebrand factor by ADAMTS13 is associated with microangiopathic acute kidney injury following trauma.
Blood Coagul Fibrinolysis
; 33(1): 14-24, 2022 Jan 01.
Artigo
Inglês
| MEDLINE | ID: mdl-34889809
14.
Resuscitation with epinephrine worsens cerebral capillary no-reflow after experimental pediatric cardiac arrest: An in vivo multiphoton microscopy evaluation.
J Cereb Blood Flow Metab
; 42(12): 2255-2269, 2022 12.
Artigo
Inglês
| MEDLINE | ID: mdl-35854408
15.
Caspase-1 is hepatoprotective during trauma and hemorrhagic shock by reducing liver injury and inflammation.
Mol Med
; 17(9-10): 1031-8, 2011.
Artigo
Inglês
| MEDLINE | ID: mdl-21666957
16.
Complement factor 3 deficiency attenuates hemorrhagic shock-related hepatic injury and systemic inflammatory response syndrome.
Am J Physiol Regul Integr Comp Physiol
; 299(5): R1175-82, 2010 Nov.
Artigo
Inglês
| MEDLINE | ID: mdl-20702808
17.
LPS Induces Active HMGB1 Release From Hepatocytes Into Exosomes Through the Coordinated Activities of TLR4 and Caspase-11/GSDMD Signaling.
Front Immunol
; 11: 229, 2020.
Artigo
Inglês
| MEDLINE | ID: mdl-32328059
18.
Cerebral microcirculatory alterations and the no-reflow phenomenon in vivo after experimental pediatric cardiac arrest.
J Cereb Blood Flow Metab
; 39(5): 913-925, 2019 05.
Artigo
Inglês
| MEDLINE | ID: mdl-29192562
19.
Changes in FADD levels, distribution, and phosphorylation in TNFalpha-induced apoptosis in hepatocytes is caspase-3, caspase-8 and BID dependent.
Apoptosis
; 13(8): 983-92, 2008 Aug.
Artigo
Inglês
| MEDLINE | ID: mdl-18543108
20.
Carbon monoxide decreases the level of iNOS protein and active dimer in IL-1beta-stimulated hepatocytes.
Nitric Oxide
; 18(4): 256-65, 2008 Jun.
Artigo
Inglês
| MEDLINE | ID: mdl-18313411