Detalhe da pesquisa
1.
A nonlinear meccano for Alzheimer's emergence by amyloid ß-mediated glutamatergic hyperactivity.
Neurobiol Dis
; 194: 106473, 2024 May.
Artigo
Inglês
| MEDLINE | ID: mdl-38493903
2.
Sephin1 Protects Neurons against Excitotoxicity Independently of the Integrated Stress Response.
Int J Mol Sci
; 21(17)2020 Aug 24.
Artigo
Inglês
| MEDLINE | ID: mdl-32846985
3.
Corrigendum to "Contribution of Neurons and Glial Cells to Complement-Mediated Synapse Removal during Development, Aging and in Alzheimer's Disease".
Mediators Inflamm
; 2019: 7539620, 2019.
Artigo
Inglês
| MEDLINE | ID: mdl-30837797
4.
Contribution of Neurons and Glial Cells to Complement-Mediated Synapse Removal during Development, Aging and in Alzheimer's Disease.
Mediators Inflamm
; 2018: 2530414, 2018.
Artigo
Inglês
| MEDLINE | ID: mdl-30533998
5.
A Neuron, Microglia, and Astrocyte Triple Co-culture Model to Study Alzheimer's Disease.
Front Aging Neurosci
; 14: 844534, 2022.
Artigo
Inglês
| MEDLINE | ID: mdl-35493929
6.
Amyloid ß / PKC-dependent alterations in NMDA receptor composition are detected in early stages of Alzheimer´s disease.
Cell Death Dis
; 13(3): 253, 2022 03 19.
Artigo
Inglês
| MEDLINE | ID: mdl-35306512