From Family Violence to Dating Violence: Testing a Dual Pathway Model.

Risk factors for adolescent perpetration of or victimization by dating violence stem from different levels of adolescents' social ecologies, including the family, individual, and peer domains. However, these multiple risk factors have not been fully integrated into a single comprehensive model of dating violence development. The present study examined prospective links between exposure to family violence in pre-adolescence; pro-violent beliefs, aggression, deviant peer affiliation, and aggression toward opposite-sex peers in early adolescence and dating violence in late adolescence. Using a longitudinal study of 461 youth (51 % female; 80 % African American, 19 % Caucasian, 1 % other ethnicities), path modeling evaluated a theoretically developed dual pathway model involving a general violence pathway and an early romantic aggression pathway. Each pathway links exposure to family violence in pre-adolescence with early adolescent pro-violent beliefs and/or aggressive behavior. In both pathways, pro-violent beliefs may reinforce aggressive behaviors between same-sex and opposite-sex peers, as well as strengthen bonds with deviant peers. In the last part of both pathways, aggressive behavior and peer deviance in early adolescence may contribute directly to late adolescent dating violence perpetration and victimization. The findings provided support for both pathways, as well as sex differences in the model.

Uric Acid Excretion Predicts Increased Blood Pressure Among American Adolescents of African Descent.

BACKGROUND: Hyperuricemia predicts the incidence of hypertension in adults and its treatment has blood pressure (BP)-lowering effects in adolescents. To date, no studies have examined the predictive usage of hyperuricemia or urinary uric acid excretion on BP changes in adolescents. Mechanistic models suggest that uric acid impairs both endothelial function and vascular compliance, which would potentially exacerbate a myriad of hypertensive mechanisms, yet little is known about interaction of uric acid and other hypertension risk factors. MATERIALS AND METHODS: The primary study was aimed at the effects of stress on BP in adolescents. A community sample of 84 low-income, urban adolescents (50% male, 95% African American, mean age = 13.36 ± 1 years) was recruited from public schools. Youth completed a 12-hour (overnight) urine collection at home and their BP was measured during rest and in response to acute psychosocial stress. Seventy-six of the adolescents participated in a follow-up visit at 1.5 years when their resting BP was reassessed. In this substudy, we assessed the relationship of renal urate excretion and BP reactivity. RESULTS: After adjusting for resting BP levels at baseline and other covariates, higher levels of uric acid excretion predicted greater BP reactivity to acute psychosocial stress and higher resting BP at 18 months. CONCLUSIONS: Urinary excretion of uric acid can serve as an alternative, noninvasive measure of serum uric acid levels that are predictive of BP changes. As hyperuricemia-associated hypertension is treatable, urban adolescents may benefit from routine screening for hyperuricemia or high uric acid excretion.