Detalhe da pesquisa
1.
Glycolysis-Mediated Activation of v-ATPase by Nicotinamide Mononucleotide Ameliorates Lipid-Induced Cardiomyopathy by Repressing the CD36-TLR4 Axis.
Circ Res
; 134(5): 505-525, 2024 03.
Artigo
Inglês
| MEDLINE | ID: mdl-38422177
2.
CD36 (SR-B2) as master regulator of cellular fatty acid homeostasis.
Curr Opin Lipidol
; 33(2): 103-111, 2022 04 01.
Artigo
Inglês
| MEDLINE | ID: mdl-35125400
3.
Guidelines on models of diabetic heart disease.
Am J Physiol Heart Circ Physiol
; 323(1): H176-H200, 2022 07 01.
Artigo
Inglês
| MEDLINE | ID: mdl-35657616
4.
Evaluation of the Interaction of Sex Hormones and Cardiovascular Function and Health.
Curr Heart Fail Rep
; 19(4): 200-212, 2022 08.
Artigo
Inglês
| MEDLINE | ID: mdl-35624387
5.
Ketone Body Exposure of Cardiomyocytes Impairs Insulin Sensitivity and Contractile Function through Vacuolar-Type H+-ATPase Disassembly-Rescue by Specific Amino Acid Supplementation.
Int J Mol Sci
; 23(21)2022 Oct 26.
Artigo
Inglês
| MEDLINE | ID: mdl-36361698
6.
Metabolic Interventions to Prevent Hypertrophy-Induced Alterations in Contractile Properties In Vitro.
Int J Mol Sci
; 22(7)2021 Mar 31.
Artigo
Inglês
| MEDLINE | ID: mdl-33807195
7.
Endurance-Type Exercise Increases Bulk and Individual Mitochondrial Protein Synthesis Rates in Rats.
Int J Sport Nutr Exerc Metab
; 30(2): 153164, 2020 03 01.
Artigo
Inglês
| MEDLINE | ID: mdl-32035417
8.
Putative Role of Protein Palmitoylation in Cardiac Lipid-Induced Insulin Resistance.
Int J Mol Sci
; 21(24)2020 Dec 11.
Artigo
Inglês
| MEDLINE | ID: mdl-33322406
9.
Augmenting Vacuolar H+-ATPase Function Prevents Cardiomyocytes from Lipid-Overload Induced Dysfunction.
Int J Mol Sci
; 21(4)2020 Feb 23.
Artigo
Inglês
| MEDLINE | ID: mdl-32102213
10.
Statins Promote Cardiac Infarct Healing by Modulating Endothelial Barrier Function Revealed by Contrast-Enhanced Magnetic Resonance Imaging.
Arterioscler Thromb Vasc Biol
; 38(1): 186-194, 2018 01.
Artigo
Inglês
| MEDLINE | ID: mdl-29146749
11.
2-Arachidonoylglycerol ameliorates inflammatory stress-induced insulin resistance in cardiomyocytes.
J Biol Chem
; 292(17): 7105-7114, 2017 04 28.
Artigo
Inglês
| MEDLINE | ID: mdl-28320859
12.
Human embryonic stem cell-derived cardiomyocytes as an in vitro model to study cardiac insulin resistance.
Biochim Biophys Acta Mol Basis Dis
; 1864(5 Pt B): 1960-1967, 2018 May.
Artigo
Inglês
| MEDLINE | ID: mdl-29277329
13.
A new leptin-mediated mechanism for stimulating fatty acid oxidation: a pivotal role for sarcolemmal FAT/CD36.
Biochem J
; 474(1): 149-162, 2017 01 01.
Artigo
Inglês
| MEDLINE | ID: mdl-27827305
14.
Regulation of the subcellular trafficking of CD36, a major determinant of cardiac fatty acid utilization.
Biochim Biophys Acta
; 1861(10): 1461-71, 2016 10.
Artigo
Inglês
| MEDLINE | ID: mdl-27090938
15.
Post-translational modifications of CD36 (SR-B2): Implications for regulation of myocellular fatty acid uptake.
Biochim Biophys Acta
; 1862(12): 2253-2258, 2016 12.
Artigo
Inglês
| MEDLINE | ID: mdl-27615427
16.
Dietary nitrate does not reduce oxygen cost of exercise or improve muscle mitochondrial function in patients with mitochondrial myopathy.
Am J Physiol Regul Integr Comp Physiol
; 312(5): R689-R701, 2017 05 01.
Artigo
Inglês
| MEDLINE | ID: mdl-28179228
17.
Preservation of myocardial fatty acid oxidation prevents diastolic dysfunction in mice subjected to angiotensin II infusion.
J Mol Cell Cardiol
; 100: 64-71, 2016 Nov.
Artigo
Inglês
| MEDLINE | ID: mdl-27693463
18.
Cardiac diastolic dysfunction in high-fat diet fed mice is associated with lipotoxicity without impairment of cardiac energetics in vivo.
Biochim Biophys Acta
; 1842(10): 1525-37, 2014 Oct.
Artigo
Inglês
| MEDLINE | ID: mdl-25086219
19.
Augmenting muscle diacylglycerol and triacylglycerol content by blocking fatty acid oxidation does not impede insulin sensitivity.
Proc Natl Acad Sci U S A
; 109(29): 11711-6, 2012 Jul 17.
Artigo
Inglês
| MEDLINE | ID: mdl-22753483
20.
Lack of UCP3 does not affect skeletal muscle mitochondrial function under lipid-challenged conditions, but leads to sudden cardiac death.
Basic Res Cardiol
; 109(6): 447, 2014.
Artigo
Inglês
| MEDLINE | ID: mdl-25344084