Detalhe da pesquisa
1.
Oxidized phospholipids are proinflammatory and proatherogenic in hypercholesterolaemic mice.
Nature
; 558(7709): 301-306, 2018 06.
Artigo
Inglês
| MEDLINE | ID: mdl-29875409
2.
Publisher Correction: Oxidized phospholipids are proinflammatory and proatherogenic in hypercholesterolaemic mice.
Nature
; 561(7724): E43, 2018 09.
Artigo
Inglês
| MEDLINE | ID: mdl-30013121
3.
PRDM16 Is a Compact Myocardium-Enriched Transcription Factor Required to Maintain Compact Myocardial Cardiomyocyte Identity in Left Ventricle.
Circulation
; 145(8): 586-602, 2022 02 22.
Artigo
Inglês
| MEDLINE | ID: mdl-34915728
4.
Cardiomyocyte Expression of ZO-1 Is Essential for Normal Atrioventricular Conduction but Does Not Alter Ventricular Function.
Circ Res
; 127(2): 284-297, 2020 07 03.
Artigo
Inglês
| MEDLINE | ID: mdl-32345129
5.
Suppression of Endothelial AGO1 Promotes Adipose Tissue Browning and Improves Metabolic Dysfunction.
Circulation
; 142(4): 365-379, 2020 07 28.
Artigo
Inglês
| MEDLINE | ID: mdl-32393053
6.
Nexilin Is a New Component of Junctional Membrane Complexes Required for Cardiac T-Tubule Formation.
Circulation
; 140(1): 55-66, 2019 07 02.
Artigo
Inglês
| MEDLINE | ID: mdl-30982350
7.
Infarct Fibroblasts Do Not Derive From Bone Marrow Lineages.
Circ Res
; 122(4): 583-590, 2018 02 16.
Artigo
Inglês
| MEDLINE | ID: mdl-29269349
8.
Increasing Mononuclear Diploid Cardiomyocytes by Loss of E2F Transcription Factor 7/8 Fails to Improve Cardiac Regeneration After Infarct.
Circulation
; 147(2): 183-186, 2023 01 10.
Artigo
Inglês
| MEDLINE | ID: mdl-36622904
9.
Impaired mitophagy facilitates mitochondrial damage in Danon disease.
J Mol Cell Cardiol
; 108: 86-94, 2017 07.
Artigo
Inglês
| MEDLINE | ID: mdl-28526246
10.
Mitochondrial reprogramming induced by CaMKIIδ mediates hypertrophy decompensation.
Circ Res
; 116(5): e28-39, 2015 Feb 27.
Artigo
Inglês
| MEDLINE | ID: mdl-25605649
11.
Selective Life-Long Skeletal Myofiber-Targeted VEGF Gene Ablation Impairs Exercise Capacity in Adult Mice.
J Cell Physiol
; 231(2): 505-11, 2016 Feb.
Artigo
Inglês
| MEDLINE | ID: mdl-26201683
12.
Connexin defects underlie arrhythmogenic right ventricular cardiomyopathy in a novel mouse model.
Hum Mol Genet
; 23(5): 1134-50, 2014 Mar 01.
Artigo
Inglês
| MEDLINE | ID: mdl-24108106
13.
Lmo7 is dispensable for skeletal muscle and cardiac function.
Am J Physiol Cell Physiol
; 309(7): C470-9, 2015 Oct 01.
Artigo
Inglês
| MEDLINE | ID: mdl-26157009
14.
Talin1 has unique expression versus talin 2 in the heart and modifies the hypertrophic response to pressure overload.
J Biol Chem
; 288(6): 4252-64, 2013 Feb 08.
Artigo
Inglês
| MEDLINE | ID: mdl-23266827
15.
Discoidin domain receptor 2 germline gene deletion leads to altered heart structure and function in the mouse.
Am J Physiol Heart Circ Physiol
; 307(5): H773-81, 2014 Sep 01.
Artigo
Inglês
| MEDLINE | ID: mdl-24993042
16.
Thymosin beta 4 is dispensable for murine cardiac development and function.
Circ Res
; 110(3): 456-64, 2012 Feb 03.
Artigo
Inglês
| MEDLINE | ID: mdl-22158707
17.
MicroRNA-133 controls cardiac hypertrophy.
Nat Med
; 13(5): 613-8, 2007 May.
Artigo
Inglês
| MEDLINE | ID: mdl-17468766
18.
Over-expression of DSCAM and COL6A2 cooperatively generates congenital heart defects.
PLoS Genet
; 7(11): e1002344, 2011 Nov.
Artigo
Inglês
| MEDLINE | ID: mdl-22072978
19.
Selective deletion of long but not short Cypher isoforms leads to late-onset dilated cardiomyopathy.
Hum Mol Genet
; 20(9): 1751-62, 2011 May 01.
Artigo
Inglês
| MEDLINE | ID: mdl-21303826
20.
ß1 integrin gene excision in the adult murine cardiac myocyte causes defective mechanical and signaling responses.
Am J Pathol
; 180(3): 952-962, 2012 Mar.
Artigo
Inglês
| MEDLINE | ID: mdl-22248583