Detalhe da pesquisa
1.
Atrial fibrillation and heart failure-associated remodeling of two-pore-domain potassium (K2P) channels in murine disease models: focus on TASK-1.
Basic Res Cardiol
; 113(4): 27, 2018 06 07.
Artigo
Inglês
| MEDLINE | ID: mdl-29881975
2.
Inward Rectifier K+ Currents Contribute to the Proarrhythmic Electrical Phenotype of Atria Overexpressing Cyclic Adenosine Monophosphate Response Element Modulator Isoform CREM-IbΔC-X.
J Am Heart Assoc
; 9(23): e016144, 2020 12.
Artigo
Inglês
| MEDLINE | ID: mdl-33191843
3.
HDAC (Histone Deacetylase) Inhibitor Valproic Acid Attenuates Atrial Remodeling and Delays the Onset of Atrial Fibrillation in Mice.
Circ Arrhythm Electrophysiol
; 12(3): e007071, 2019 03.
Artigo
Inglês
| MEDLINE | ID: mdl-30879335
4.
Homozygous CREM-IbΔC-X Overexpressing Mice Are a Reliable and Effective Disease Model for Atrial Fibrillation.
Front Pharmacol
; 9: 706, 2018.
Artigo
Inglês
| MEDLINE | ID: mdl-30026696
5.
Characterization of the Genetic Program Linked to the Development of Atrial Fibrillation in CREM-IbΔC-X Mice.
Circ Arrhythm Electrophysiol
; 10(8)2017 Aug.
Artigo
Inglês
| MEDLINE | ID: mdl-28784605
6.
Stretch-activated two-pore-domain (K2P) potassium channels in the heart: Focus on atrial fibrillation and heart failure.
Prog Biophys Mol Biol
; 130(Pt B): 233-243, 2017 11.
Artigo
Inglês
| MEDLINE | ID: mdl-28526353