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1.
J Biol Chem ; 286(43): 37578-89, 2011 Oct 28.
Artigo em Inglês | MEDLINE | ID: mdl-21880720

RESUMO

Myeloperoxidase (MPO) is a prime candidate for promoting oxidative stress during inflammation. This abundant enzyme of neutrophils uses hydrogen peroxide to oxidize chloride to highly reactive and toxic chlorine bleach. We have identified 2-thioxanthines as potent mechanism-based inactivators of MPO. Mass spectrometry and x-ray crystal structures revealed that these inhibitors become covalently attached to the heme prosthetic groups of the enzyme. We propose a mechanism whereby 2-thioxanthines are oxidized, and their incipient free radicals react with the heme groups of the enzyme before they can exit the active site. 2-Thioxanthines inhibited MPO in plasma and decreased protein chlorination in a mouse model of peritonitis. They slowed but did not prevent neutrophils from killing bacteria and were poor inhibitors of thyroid peroxidase. Our study shows that MPO is susceptible to the free radicals it generates, and this Achilles' heel of the enzyme can be exploited to block oxidative stress during inflammation.


Assuntos
Inibidores Enzimáticos , Neutrófilos/enzimologia , Estresse Oxidativo/efeitos dos fármacos , Peritonite/enzimologia , Peroxidase , Xantinas , Animais , Cristalografia por Raios X , Modelos Animais de Doenças , Inibidores Enzimáticos/química , Inibidores Enzimáticos/farmacologia , Humanos , Inflamação/tratamento farmacológico , Inflamação/etnologia , Inflamação/microbiologia , Inflamação/patologia , Camundongos , Neutrófilos/patologia , Oxirredução/efeitos dos fármacos , Peritonite/tratamento farmacológico , Peritonite/patologia , Peroxidase/antagonistas & inibidores , Peroxidase/química , Peroxidase/metabolismo , Xantinas/química , Xantinas/farmacologia
2.
Carbohydr Res ; 342(7): 970-4, 2007 May 21.
Artigo em Inglês | MEDLINE | ID: mdl-17324385

RESUMO

The investigation of drug metabolism requires substantial amount of metabolites. Isolation from urine is tedious, therefore, the material obtained by synthesis is preferred. Substantial amounts of three tentative drug metabolites, phenolic glucuronides, have been prepared using easily available glycosyl donors. The final products [3(2-N-methyl-N-isopropylaminoethoxy)phenyl] beta-D-glucopyranosiduronic acid, 4-amino-3,5-dimethylphenyl beta-D-glucopyranosiduronic acid and [2(S)-propanoyl-6-O-naphthyl] beta-D-glucopyranuronic acid are useful as, for example, reference material in metabolite investigations.


Assuntos
Ácido Glucurônico/síntese química , Ácido Glucurônico/urina , Glicosídeos/síntese química , Glicosídeos/urina , Fenóis/metabolismo , Ácido Glucurônico/química , Glicosídeos/química , Estrutura Molecular , Fenóis/urina
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