Detalhe da pesquisa
1.
SGLT2 Inhibitors Decrease Overhydration and Proteasuria in Patients with Chronic Kidney Disease: A Longitudinal Observational Study.
Kidney Blood Press Res
; 49(1): 124-134, 2024.
Artigo
Inglês
| MEDLINE | ID: mdl-38228104
2.
Sodium retention in nephrotic syndrome is independent of the activation of the membrane-anchored serine protease prostasin (CAP1/PRSS8) and its enzymatic activity.
Pflugers Arch
; 474(6): 613-624, 2022 06.
Artigo
Inglês
| MEDLINE | ID: mdl-35312839
3.
Proteolytic activation of the epithelial sodium channel (ENaC) by factor VII activating protease (FSAP) and its relevance for sodium retention in nephrotic mice.
Pflugers Arch
; 474(2): 217-229, 2022 02.
Artigo
Inglês
| MEDLINE | ID: mdl-34870751
4.
Renal effects of the serine protease inhibitor aprotinin in healthy conscious mice.
Acta Pharmacol Sin
; 43(1): 111-120, 2022 Jan.
Artigo
Inglês
| MEDLINE | ID: mdl-33758357
5.
Experimental nephrotic syndrome leads to proteolytic activation of the epithelial Na+ channel in the mouse kidney.
Am J Physiol Renal Physiol
; 321(4): F480-F493, 2021 10 01.
Artigo
Inglês
| MEDLINE | ID: mdl-34423678
6.
Proteinuric chronic kidney disease is associated with altered red blood cell lifespan, deformability and metabolism.
Kidney Int
; 100(6): 1227-1239, 2021 12.
Artigo
Inglês
| MEDLINE | ID: mdl-34537228
7.
Aprotinin prevents proteolytic epithelial sodium channel (ENaC) activation and volume retention in nephrotic syndrome.
Kidney Int
; 93(1): 159-172, 2018 01.
Artigo
Inglês
| MEDLINE | ID: mdl-29042083
8.
Amiloride versus furosemide for the treatment of edema in patients with nephrotic syndrome: A pilot study (AMILOR).
Acta Physiol (Oxf)
; : e14183, 2024 Jun 01.
Artigo
Inglês
| MEDLINE | ID: mdl-38822593
9.
Proteolytic Activity against the Distal Polybasic Tract of the Gamma Subunit of the Epithelial Sodium Channel ENaC in Nephrotic Urine.
Curr Med Chem
; 29(42): 6433-6445, 2022.
Artigo
Inglês
| MEDLINE | ID: mdl-35676854
10.
Proteasuria in nephrotic syndrome-quantification and proteomic profiling.
J Proteomics
; 230: 103981, 2021 01 06.
Artigo
Inglês
| MEDLINE | ID: mdl-32927112
11.
Plasminogen deficiency does not prevent sodium retention in a genetic mouse model of experimental nephrotic syndrome.
Acta Physiol (Oxf)
; 231(1): e13512, 2021 01.
Artigo
Inglês
| MEDLINE | ID: mdl-32455507
12.
Zymogen-locked mutant prostasin (Prss8) leads to incomplete proteolytic activation of the epithelial sodium channel (ENaC) and severely compromises triamterene tolerance in mice.
Acta Physiol (Oxf)
; 232(1): e13640, 2021 05.
Artigo
Inglês
| MEDLINE | ID: mdl-33650216
13.
Proteasuria-The impact of active urinary proteases on sodium retention in nephrotic syndrome.
Acta Physiol (Oxf)
; 225(4): e13249, 2019 04.
Artigo
Inglês
| MEDLINE | ID: mdl-30597733
14.
Urokinase-type plasminogen activator (uPA) is not essential for epithelial sodium channel (ENaC)-mediated sodium retention in experimental nephrotic syndrome.
Acta Physiol (Oxf)
; 227(4): e13286, 2019 12.
Artigo
Inglês
| MEDLINE | ID: mdl-31006168