Detalhe da pesquisa
1.
Induced Pluripotent Stem Cell (iPSC)-Derived Extracellular Vesicles Are Safer and More Effective for Cardiac Repair Than iPSCs.
Circ Res
; 122(2): 296-309, 2018 01 19.
Artigo
Inglês
| MEDLINE | ID: mdl-29118058
2.
Deletion of Interleukin-6 Attenuates Pressure Overload-Induced Left Ventricular Hypertrophy and Dysfunction.
Circ Res
; 118(12): 1918-1929, 2016 06 10.
Artigo
Inglês
| MEDLINE | ID: mdl-27126808
3.
Carbon monoxide induces a late preconditioning-mimetic cardioprotective and antiapoptotic milieu in the myocardium.
J Mol Cell Cardiol
; 52(1): 228-36, 2012 Jan.
Artigo
Inglês
| MEDLINE | ID: mdl-22119801
4.
A murine model of inducible, cardiac-specific deletion of STAT3: its use to determine the role of STAT3 in the upregulation of cardioprotective proteins by ischemic preconditioning.
J Mol Cell Cardiol
; 50(4): 589-97, 2011 Apr.
Artigo
Inglês
| MEDLINE | ID: mdl-21223971
5.
Endothelial nitric oxide synthase plays an obligatory role in the late phase of ischemic preconditioning by activating the protein kinase C epsilon p44/42 mitogen-activated protein kinase pSer-signal transducers and activators of transcription1/3 pathway.
Circulation
; 116(5): 535-44, 2007 Jul 31.
Artigo
Inglês
| MEDLINE | ID: mdl-17606840
6.
Role of the protein kinase C-epsilon-Raf-1-MEK-1/2-p44/42 MAPK signaling cascade in the activation of signal transducers and activators of transcription 1 and 3 and induction of cyclooxygenase-2 after ischemic preconditioning.
Circulation
; 112(13): 1971-8, 2005 Sep 27.
Artigo
Inglês
| MEDLINE | ID: mdl-16172266
7.
Protein kinase Cepsilon interacts with and inhibits the permeability transition pore in cardiac mitochondria.
Circ Res
; 92(8): 873-80, 2003 May 02.
Artigo
Inglês
| MEDLINE | ID: mdl-12663490
8.
The late phase of ischemic preconditioning induces a prosurvival genetic program that results in marked attenuation of apoptosis.
J Mol Cell Cardiol
; 42(6): 1075-85, 2007 Jun.
Artigo
Inglês
| MEDLINE | ID: mdl-17490677
9.
Tumor necrosis factor-alpha does not modulate ischemia/reperfusion injury in naïve myocardium but is essential for the development of late preconditioning.
J Mol Cell Cardiol
; 37(1): 51-61, 2004 Jul.
Artigo
Inglês
| MEDLINE | ID: mdl-15242735