Estatinas nas Síndromes Coronárias Agudas

Aarão, Amanda Rezende; Sposito, Alexandre Russo; Sousa, Francisco Thiago Tomaz de; Aguiar Filho, Gentil Barreira.
São Paulo; IDPC; 2009. 3696 p.
Monografia Português | LILACS, SES-SP, SES SP - Instituto Dante Pazzanese de Cardiologia, SES-SP | ID: biblio-1076095

Documentos relacionados

  1. Small LDL subfractions are associated with coronary atherosclerosis despite no differences in conventional lipids.
  2. Complex effects of sequence variants on lipid levels and coronary artery disease.
  3. Remnants and Triglyceride-Rich Lipoproteins in Prevention of Premature Coronary Heart Disease.
  4. Increased absorption of phytosterols is the simplest and most plausible explanation for coronary artery disease risk not accounted for by non-HDL cholesterol in high cholesterol absorbers.
  5. Effect of Novel Stratified Lipid Risk by "LDL-Window" and Flow-Mediated Dilation on the Prognosis of Coronary Artery Disease Using the FMD-J Study A Data.
  6. Genetic determinants of clinical heterogeneity of the coronary artery disease in the population of Hyderabad, India.
  7. Elevated postprandial triglyceride-rich lipoproteins in patients with diabetes and stable coronary artery disease correlated with early renal damage and systemic inflammation.
  8. Variability in atherogenic lipoproteins and coronary artery disease progression.
  9. Apolipoprotein A-V is a potential target for treating coronary artery disease: evidence from genetic and metabolomic analyses.
  10. Effect of lipoprotein <b>&#945;</b> on coronary stents expansion and its risk factors.