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Phospholipid transfer protein (PLTP) deficiency accelerates memory dysfunction through altering amyloid precursor protein (APP) processing in a mouse model of Alzheimer's disease.
Tong, Yawei; Sun, Yang; Tian, Xiaosheng; Zhou, Ting; Wang, Hecheng; Zhang, Tao; Zhan, Rui; Zhao, Lei; Kuerban, Bolati; Li, Zhengqian; Wang, Qiudian; Jin, Yinglan; Fan, Dongsheng; Guo, Xiangyang; Han, Hongbin; Qin, Shucun; Chui, Dehua.
  • Tong Y; Neuroscience Research Institute and Department of Neurobiology, Key Laboratory for Neuroscience, Ministry of Education and Ministry of Public Health, Health Science Center, Peking University, Beijing, China.
  • Sun Y; Neuroscience Research Institute and Department of Neurobiology, Key Laboratory for Neuroscience, Ministry of Education and Ministry of Public Health, Health Science Center, Peking University, Beijing, China.
  • Tian X; Neuroscience Research Institute and Department of Neurobiology, Key Laboratory for Neuroscience, Ministry of Education and Ministry of Public Health, Health Science Center, Peking University, Beijing, China.
  • Zhou T; Neuroscience Research Institute and Department of Neurobiology, Key Laboratory for Neuroscience, Ministry of Education and Ministry of Public Health, Health Science Center, Peking University, Beijing, China.
  • Wang H; Neuroscience Research Institute and Department of Neurobiology, Key Laboratory for Neuroscience, Ministry of Education and Ministry of Public Health, Health Science Center, Peking University, Beijing, China.
  • Zhang T; Neuroscience Research Institute and Department of Neurobiology, Key Laboratory for Neuroscience, Ministry of Education and Ministry of Public Health, Health Science Center, Peking University, Beijing, China.
  • Zhan R; Neuroscience Research Institute and Department of Neurobiology, Key Laboratory for Neuroscience, Ministry of Education and Ministry of Public Health, Health Science Center, Peking University, Beijing, China.
  • Zhao L; Neuroscience Research Institute and Department of Neurobiology, Key Laboratory for Neuroscience, Ministry of Education and Ministry of Public Health, Health Science Center, Peking University, Beijing, China.
  • Kuerban B; Neuroscience Research Institute and Department of Neurobiology, Key Laboratory for Neuroscience, Ministry of Education and Ministry of Public Health, Health Science Center, Peking University, Beijing, China.
  • Li Z; Department of Anesthesiology, Peking University Third Hospital, Beijing, China.
  • Wang Q; Neuroscience Research Institute and Department of Neurobiology, Key Laboratory for Neuroscience, Ministry of Education and Ministry of Public Health, Health Science Center, Peking University, Beijing, China.
  • Jin Y; Neuroscience Research Institute and Department of Neurobiology, Key Laboratory for Neuroscience, Ministry of Education and Ministry of Public Health, Health Science Center, Peking University, Beijing, China.
  • Fan D; Department of Neurology, Peking University Third Hospital, Beijing, China dchui@bjmu.edu.cn dsfan@sina.com hanhongbin@bjmu.edu.cn sucunqin@hotmail.com.
  • Guo X; Department of Anesthesiology, Peking University Third Hospital, Beijing, China.
  • Han H; Department of Radiology, Peking University Third Hospital, Beijing, China dchui@bjmu.edu.cn dsfan@sina.com hanhongbin@bjmu.edu.cn sucunqin@hotmail.com.
  • Qin S; Key Laboratory of Atherosclerosis in Universities of Shandong, Institute of Atherosclerosis, Taishan Medical University, Taian, China, dchui@bjmu.edu.cn dsfan@sina.com hanhongbin@bjmu.edu.cn sucunqin@hotmail.com.
  • Chui D; Neuroscience Research Institute and Department of Neurobiology, Key Laboratory for Neuroscience, Ministry of Education and Ministry of Public Health, Health Science Center, Peking University, Beijing, China, Department of Neurology, Peking University Third Hospital, Beijing, China dchui@bjmu.edu.cn
Hum Mol Genet ; 24(19): 5388-403, 2015 Oct 01.
Article en En | MEDLINE | ID: mdl-26160914
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Texto completo: 1 Banco de datos: MEDLINE Asunto principal: Precursor de Proteína beta-Amiloide / Proteínas de Transferencia de Fosfolípidos / Enfermedad de Alzheimer / Trastornos de la Memoria Tipo de estudio: Prognostic_studies Límite: Animals / Humans Idioma: En Año: 2015 Tipo del documento: Article
Texto completo
Imprimir
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PubMed Links
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Texto completo: 1 Banco de datos: MEDLINE Asunto principal: Precursor de Proteína beta-Amiloide / Proteínas de Transferencia de Fosfolípidos / Enfermedad de Alzheimer / Trastornos de la Memoria Tipo de estudio: Prognostic_studies Límite: Animals / Humans Idioma: En Año: 2015 Tipo del documento: Article