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The Nuclear Factor (Erythroid-derived 2)-like 2 and Proteasome Maturation Protein Axis Mediate Bortezomib Resistance in Multiple Myeloma.
Li, Bingzong; Fu, Jinxiang; Chen, Ping; Ge, Xueping; Li, Yali; Kuiatse, Isere; Wang, Hua; Wang, Huihan; Zhang, Xingding; Orlowski, Robert Z.
  • Li B; From the Department of Hematology, Second Affiliated Hospital of Soochow University, Suzhou 215006, Jiangsu, China and the Departments of Lymphoma and Myeloma and.
  • Fu J; From the Department of Hematology, Second Affiliated Hospital of Soochow University, Suzhou 215006, Jiangsu, China and.
  • Chen P; From the Department of Hematology, Second Affiliated Hospital of Soochow University, Suzhou 215006, Jiangsu, China and.
  • Ge X; From the Department of Hematology, Second Affiliated Hospital of Soochow University, Suzhou 215006, Jiangsu, China and.
  • Li Y; From the Department of Hematology, Second Affiliated Hospital of Soochow University, Suzhou 215006, Jiangsu, China and.
  • Kuiatse I; the Departments of Lymphoma and Myeloma and.
  • Wang H; the Departments of Lymphoma and Myeloma and.
  • Wang H; the Departments of Lymphoma and Myeloma and.
  • Zhang X; the Departments of Lymphoma and Myeloma and.
  • Orlowski RZ; the Departments of Lymphoma and Myeloma and Experimental Therapeutics, The University of Texas MD Anderson Cancer Center, Houston, Texas 77030 rorlowsk@mdanderson.org.
J Biol Chem ; 290(50): 29854-68, 2015 Dec 11.
Article en En | MEDLINE | ID: mdl-26483548
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Texto completo: 1 Banco de datos: MEDLINE Asunto principal: Chaperonas Moleculares / Resistencia a Antineoplásicos / Factor 2 Relacionado con NF-E2 / Bortezomib / Mieloma Múltiple Tipo de estudio: Prognostic_studies Límite: Humans Idioma: En Año: 2015 Tipo del documento: Article
Texto completo
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PubMed Links
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Texto completo: 1 Banco de datos: MEDLINE Asunto principal: Chaperonas Moleculares / Resistencia a Antineoplásicos / Factor 2 Relacionado con NF-E2 / Bortezomib / Mieloma Múltiple Tipo de estudio: Prognostic_studies Límite: Humans Idioma: En Año: 2015 Tipo del documento: Article