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Non-prion-type transmission in A53T α-synuclein transgenic mice: a normal component of spinal homogenates from naïve non-transgenic mice induces robust α-synuclein pathology.
Sacino, Amanda N; Ayers, Jacob I; Brooks, Mieu M T; Chakrabarty, Paramita; Hudson, Vincent J; Howard, Jasie K; Golde, Todd E; Giasson, Benoit I; Borchelt, David R.
  • Sacino AN; Department of Neuroscience, College of Medicine University of Florida, Gainesville, FL, 32610, USA.
  • Ayers JI; Center for Translational Research in Neurodegenerative Disease, College of Medicine University of Florida, Gainesville, FL, 32610, USA.
  • Brooks MM; Department of Neuroscience, College of Medicine University of Florida, Gainesville, FL, 32610, USA.
  • Chakrabarty P; Center for Translational Research in Neurodegenerative Disease, College of Medicine University of Florida, Gainesville, FL, 32610, USA.
  • Hudson VJ; McKnight Brain Institute, College of Medicine University of Florida, Gainesville, FL, 32610, USA.
  • Howard JK; Department of Neuroscience, College of Medicine University of Florida, Gainesville, FL, 32610, USA.
  • Golde TE; Center for Translational Research in Neurodegenerative Disease, College of Medicine University of Florida, Gainesville, FL, 32610, USA.
  • Giasson BI; Department of Neuroscience, College of Medicine University of Florida, Gainesville, FL, 32610, USA.
  • Borchelt DR; Center for Translational Research in Neurodegenerative Disease, College of Medicine University of Florida, Gainesville, FL, 32610, USA.
Acta Neuropathol ; 131(1): 151-4, 2016 Jan.
Article en En | MEDLINE | ID: mdl-26541429

Texto completo: 1 Banco de datos: MEDLINE Asunto principal: Priones / Alfa-Sinucleína Límite: Animals / Humans Idioma: En Año: 2016 Tipo del documento: Article

Texto completo: 1 Banco de datos: MEDLINE Asunto principal: Priones / Alfa-Sinucleína Límite: Animals / Humans Idioma: En Año: 2016 Tipo del documento: Article