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Mitochondrial oxidative stress-induced brain and hippocampus apoptosis decrease through modulation of caspase activity, Ca2+ influx and inflammatory cytokine molecular pathways in the docetaxel-treated mice by melatonin and selenium treatments.
Ataizi, Zeki Serdar; Ertilav, Kemal; Naziroglu, Mustafa.
  • Ataizi ZS; Departmant of Neurosurgery, Yunus Emre General State Hospital, Eskisehir, Turkey.
  • Ertilav K; Departmant of Neurosurgery, Faculty of Medicine, Suleyman Demirel University, Isparta, Turkey.
  • Naziroglu M; Neuroscience Research Center, Suleyman Demirel University, Isparta, Turkey. mustafanaziroglu@sdu.edu.tr.
Metab Brain Dis ; 34(4): 1077-1089, 2019 08.
Article en En | MEDLINE | ID: mdl-31197678

Texto completo: 1 Banco de datos: MEDLINE Asunto principal: Selenio / Encéfalo / Apoptosis / Estrés Oxidativo / Fármacos Neuroprotectores / Docetaxel / Melatonina / Mitocondrias Límite: Animals Idioma: En Año: 2019 Tipo del documento: Article

Texto completo: 1 Banco de datos: MEDLINE Asunto principal: Selenio / Encéfalo / Apoptosis / Estrés Oxidativo / Fármacos Neuroprotectores / Docetaxel / Melatonina / Mitocondrias Límite: Animals Idioma: En Año: 2019 Tipo del documento: Article