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MMP9 inhibition increases erythropoiesis in RPS14-deficient del(5q) MDS models through suppression of TGF-ß pathways.
Youn, Minyoung; Huang, Haigen; Chen, Cheng; Kam, Sharon; Wilkes, Mark C; Chae, Hee-Don; Sridhar, Kunju J; Greenberg, Peter L; Glader, Bertil; Narla, Anupama; Lin, Shuo; Sakamoto, Kathleen M.
  • Youn M; Department of Pediatrics, Stanford University School of Medicine, Stanford, CA.
  • Huang H; Department of Molecular, Cell and Developmental Biology, University of California Los Angeles, Los Angeles, CA; and.
  • Chen C; Department of Molecular, Cell and Developmental Biology, University of California Los Angeles, Los Angeles, CA; and.
  • Kam S; Department of Pediatrics, Stanford University School of Medicine, Stanford, CA.
  • Wilkes MC; Department of Pediatrics, Stanford University School of Medicine, Stanford, CA.
  • Chae HD; Department of Pediatrics, Stanford University School of Medicine, Stanford, CA.
  • Sridhar KJ; Stanford University Cancer Center, Stanford, CA.
  • Greenberg PL; Stanford University Cancer Center, Stanford, CA.
  • Glader B; Department of Pediatrics, Stanford University School of Medicine, Stanford, CA.
  • Narla A; Department of Pediatrics, Stanford University School of Medicine, Stanford, CA.
  • Lin S; Department of Molecular, Cell and Developmental Biology, University of California Los Angeles, Los Angeles, CA; and.
  • Sakamoto KM; Department of Pediatrics, Stanford University School of Medicine, Stanford, CA.
Blood Adv ; 3(18): 2751-2763, 2019 09 24.
Article en En | MEDLINE | ID: mdl-31540902
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Texto completo: 1 Banco de datos: MEDLINE Asunto principal: Factor de Crecimiento Transformador beta / Metaloproteinasa 9 de la Matriz / Eritropoyesis Tipo de estudio: Prognostic_studies Límite: Humans Idioma: En Año: 2019 Tipo del documento: Article
Texto completo
Imprimir
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Search on Google

Texto completo: 1 Banco de datos: MEDLINE Asunto principal: Factor de Crecimiento Transformador beta / Metaloproteinasa 9 de la Matriz / Eritropoyesis Tipo de estudio: Prognostic_studies Límite: Humans Idioma: En Año: 2019 Tipo del documento: Article