PLAC1 is essential for FGF7/FGFRIIIb-induced Akt-mediated cancer cell proliferation.

Roldán, Diana Barea; Grimmler, Matthias; Hartmann, Christoph; Hubich-Rau, Stefanie; Beißert, Tim; Paret, Claudia; Cagna, Giuseppe; Rohde, Christoph; Wöll, Stefan; Koslowski, Michael; Türeci, Özlem; Sahin, Ugur.

Roldán DB; TRON-Translational Oncology at the University Medical Center of the Johannes Gutenberg University Mainz, Mainz, Germany.
Grimmler M; These authors contributed equally to this work.
Hartmann C; Formerly of TRON-Translational Oncology at the University Medical Center of the Johannes Gutenberg University Mainz, Mainz, Germany.
Hubich-Rau S; Current address: DiaSys Diagnostic Systems GmbH, Holzheim, Germany.
Beißert T; These authors contributed equally to this work.
Paret C; Formerly of TRON-Translational Oncology at the University Medical Center of the Johannes Gutenberg University Mainz, Mainz, Germany.
Cagna G; Current address: Merck KGaA, Darmstadt, Germany.
Rohde C; These authors contributed equally to this work.
Wöll S; TRON-Translational Oncology at the University Medical Center of the Johannes Gutenberg University Mainz, Mainz, Germany.
Koslowski M; These authors contributed equally to this work.
Türeci Ö; TRON-Translational Oncology at the University Medical Center of the Johannes Gutenberg University Mainz, Mainz, Germany.
Sahin U; Formerly of TRON-Translational Oncology at the University Medical Center of the Johannes Gutenberg University Mainz, Mainz, Germany.