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Suppression of canonical TGF-ß signaling enables GATA4 to interact with H3K27me3 demethylase JMJD3 to promote cardiomyogenesis.
Riching, Andrew S; Danis, Etienne; Zhao, Yuanbiao; Cao, Yingqiong; Chi, Congwu; Bagchi, Rushita A; Klein, Brianna J; Xu, Hongyan; Kutateladze, Tatiana G; McKinsey, Timothy A; Buttrick, Peter M; Song, Kunhua.
  • Riching AS; Division of Cardiology, Department of Medicine, University of Colorado Anschutz Medical Campus, Aurora, CO 80045, USA; Gates Center for Regenerative Medicine and Stem Cell Biology, University of Colorado Anschutz Medical Campus, Aurora, CO 80045, USA; The Consortium for Fibrosis Research & Trans
  • Danis E; Department of Pediatrics, University of Colorado Anschutz Medical Campus, Aurora, CO 80045, USA.
  • Zhao Y; Division of Cardiology, Department of Medicine, University of Colorado Anschutz Medical Campus, Aurora, CO 80045, USA; Gates Center for Regenerative Medicine and Stem Cell Biology, University of Colorado Anschutz Medical Campus, Aurora, CO 80045, USA; The Consortium for Fibrosis Research & Trans
  • Cao Y; Division of Cardiology, Department of Medicine, University of Colorado Anschutz Medical Campus, Aurora, CO 80045, USA; Gates Center for Regenerative Medicine and Stem Cell Biology, University of Colorado Anschutz Medical Campus, Aurora, CO 80045, USA; The Consortium for Fibrosis Research & Trans
  • Chi C; Division of Cardiology, Department of Medicine, University of Colorado Anschutz Medical Campus, Aurora, CO 80045, USA; Gates Center for Regenerative Medicine and Stem Cell Biology, University of Colorado Anschutz Medical Campus, Aurora, CO 80045, USA; The Consortium for Fibrosis Research & Trans
  • Bagchi RA; Division of Cardiology, Department of Medicine, University of Colorado Anschutz Medical Campus, Aurora, CO 80045, USA; The Consortium for Fibrosis Research & Translation, University of Colorado Anschutz Medical Campus, Aurora, CO 80045, USA.
  • Klein BJ; Department of Pharmacology, University of Colorado Anschutz Medical Campus, Aurora, CO 80045, USA.
  • Xu H; Department of Population Health Sciences, Medical College of Georgia, Augusta University, Augusta, GA 30912, USA.
  • Kutateladze TG; Pharmacology Graduate Program, University of Colorado Anschutz Medical Campus, Aurora, CO 80045, USA; Department of Pharmacology, University of Colorado Anschutz Medical Campus, Aurora, CO 80045, USA.
  • McKinsey TA; Division of Cardiology, Department of Medicine, University of Colorado Anschutz Medical Campus, Aurora, CO 80045, USA; The Consortium for Fibrosis Research & Translation, University of Colorado Anschutz Medical Campus, Aurora, CO 80045, USA; Pharmacology Graduate Program, University of Colorado
  • Buttrick PM; Division of Cardiology, Department of Medicine, University of Colorado Anschutz Medical Campus, Aurora, CO 80045, USA.
  • Song K; Division of Cardiology, Department of Medicine, University of Colorado Anschutz Medical Campus, Aurora, CO 80045, USA; Gates Center for Regenerative Medicine and Stem Cell Biology, University of Colorado Anschutz Medical Campus, Aurora, CO 80045, USA; The Consortium for Fibrosis Research & Trans
J Mol Cell Cardiol ; 153: 44-59, 2021 04.
Article en En | MEDLINE | ID: mdl-33359755
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Texto completo: 1 Banco de datos: MEDLINE Asunto principal: Factor de Crecimiento Transformador beta / Regulación del Desarrollo de la Expresión Génica / Miocitos Cardíacos / Factor de Transcripción GATA4 / Células Madre Pluripotentes Inducidas / Histona Demetilasas con Dominio de Jumonji Tipo de estudio: Prognostic_studies Límite: Animals / Humans Idioma: En Año: 2021 Tipo del documento: Article
Texto completo
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PubMed Links
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Texto completo: 1 Banco de datos: MEDLINE Asunto principal: Factor de Crecimiento Transformador beta / Regulación del Desarrollo de la Expresión Génica / Miocitos Cardíacos / Factor de Transcripción GATA4 / Células Madre Pluripotentes Inducidas / Histona Demetilasas con Dominio de Jumonji Tipo de estudio: Prognostic_studies Límite: Animals / Humans Idioma: En Año: 2021 Tipo del documento: Article