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CHIP-associated mutant ASXL1 in blood cells promotes solid tumor progression.
Liu, Xiaoxiao; Sato, Naru; Shimosato, Yuko; Wang, Teh-Wei; Denda, Tamami; Chang, Yu-Hsuan; Yabushita, Tomohiro; Fujino, Takeshi; Asada, Shuhei; Tanaka, Yosuke; Fukuyama, Tomofusa; Enomoto, Yutaka; Ota, Yasunori; Sakamoto, Takeharu; Kitamura, Toshio; Goyama, Susumu.
  • Liu X; Division of Molecular Oncology, Department of Computational Biology and Medical Sciences, Graduate School of Frontier Sciences, The University of Tokyo, Tokyo, Japan.
  • Sato N; Division of Cellular Therapy, The Institute of Medical Science, The University of Tokyo, Tokyo, Japan.
  • Shimosato Y; Division of Cellular Therapy, The Institute of Medical Science, The University of Tokyo, Tokyo, Japan.
  • Wang TW; Division of Cancer Cell Biology, The Institute of Medical Science, The University of Tokyo, Tokyo, Japan.
  • Denda T; Department of Pathology, The Institute of Medical Science Research Hospital, The University of Tokyo, Tokyo, Japan.
  • Chang YH; Division of Cellular Therapy, The Institute of Medical Science, The University of Tokyo, Tokyo, Japan.
  • Yabushita T; Division of Cellular Therapy, The Institute of Medical Science, The University of Tokyo, Tokyo, Japan.
  • Fujino T; Division of Cellular Therapy, The Institute of Medical Science, The University of Tokyo, Tokyo, Japan.
  • Asada S; Division of Cellular Therapy, The Institute of Medical Science, The University of Tokyo, Tokyo, Japan.
  • Tanaka Y; The Institute of Laboratory Animals, Tokyo Women's Medical University, Tokyo, Japan.
  • Fukuyama T; Division of Cellular Therapy, The Institute of Medical Science, The University of Tokyo, Tokyo, Japan.
  • Enomoto Y; Division of Cellular Therapy, The Institute of Medical Science, The University of Tokyo, Tokyo, Japan.
  • Ota Y; Division of Cellular Therapy, The Institute of Medical Science, The University of Tokyo, Tokyo, Japan.
  • Sakamoto T; Department of Pathology, The Institute of Medical Science Research Hospital, The University of Tokyo, Tokyo, Japan.
  • Kitamura T; Department of Cancer Biology, Institute of Biomedical Science, Kansai Medical University, Osaka, Japan.
  • Goyama S; Division of Cellular Therapy, The Institute of Medical Science, The University of Tokyo, Tokyo, Japan.
Cancer Sci ; 113(4): 1182-1194, 2022 Apr.
Article en En | MEDLINE | ID: mdl-35133065
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Texto completo: 1 Banco de datos: MEDLINE Asunto principal: Proteínas Represoras / Hematopoyesis Clonal / Neoplasias Tipo de estudio: Prognostic_studies / Risk_factors_studies Límite: Animals Idioma: En Año: 2022 Tipo del documento: Article
Texto completo
Imprimir
XML
PubMed Links
Search on Google

Texto completo: 1 Banco de datos: MEDLINE Asunto principal: Proteínas Represoras / Hematopoyesis Clonal / Neoplasias Tipo de estudio: Prognostic_studies / Risk_factors_studies Límite: Animals Idioma: En Año: 2022 Tipo del documento: Article