Exogenous IL-10 posttreatment along with TLR4 and TNFR1 blockade improves tissue antioxidant status by modulating sepsis-induced macrophage polarization.

Sawoo, Ritasha; Dey, Rajen; Ghosh, Rituparna; Bishayi, Biswadev

Sawoo, Ritasha; Dey, Rajen; Ghosh, Rituparna; Bishayi, Biswadev.

Sawoo R; Department of Physiology, Immunology Laboratory, University of Calcutta, University Colleges of Science and Technology, Calcutta, India.
Dey R; Department of Physiology, Immunology Laboratory, University of Calcutta, University Colleges of Science and Technology, Calcutta, India.
Ghosh R; Department of Physiology, Immunology Laboratory, University of Calcutta, University Colleges of Science and Technology, Calcutta, India.
Bishayi B; Department of Physiology, Immunology Laboratory, University of Calcutta, University Colleges of Science and Technology, Calcutta, India.

J Appl Toxicol ; 43(10): 1549-1572, 2023 10.

Article en En | MEDLINE | ID: mdl-37177863

Asunto(s)

Antioxidantes; Sepsis; Masculino; Ratones; Animales; Antioxidantes/farmacología; Antioxidantes/uso terapéutico; Antioxidantes/metabolismo; Interleucina-10; Receptor Toll-Like 4/metabolismo; Lipopolisacáridos/toxicidad; Sepsis/tratamiento farmacológico; Sepsis/metabolismo; Macrófagos; Factor de Necrosis Tumoral alfa/metabolismo; Antiinflamatorios/metabolismo; Antiinflamatorios/farmacología; Antiinflamatorios/uso terapéutico

Palabras clave

IL-10; TLR2; TLR4; antioxidants; inflammation; macrophage polarization

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Texto completo: 1 Banco de datos: MEDLINE Asunto principal: Sepsis / Antioxidantes Tipo de estudio: Prognostic_studies Límite: Animals Idioma: En Año: 2023 Tipo del documento: Article

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