H. pylori-encoded CagA disrupts tight junctions and induces invasiveness of AGS gastric carcinoma cells via Cdx2-dependent targeting of Claudin-2.
Cell Immunol
; 286(1-2): 22-30, 2013.
Article
em En
| MEDLINE
| ID: mdl-24287273
Palavras-chave
AJC; CagA; Cdx2; Claudin-2; DS; EDTA; EMSA; FCS; HNF; Helicobacter pylori; IBD; Invasiveness; PI; Signal transduction; TJ; TJ zone; TJz; Tight junction; ZO-1; apical junctional complex; cytotoxin-associated gene A; desmosome; electrophoretic mobility shift assay; ethylenediaminetetraacetic acid disodium salt; fetal calf serum; hepatocyte nuclear factor; inflammatory bowel disease; propidium iodide; tight junction; zonulae occludentes-1
Texto completo:
1
Base de dados:
MEDLINE
Assunto principal:
Neoplasias Gástricas
/
Proteínas de Bactérias
/
Adenocarcinoma
/
Helicobacter pylori
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Proteínas de Homeodomínio
/
Junções Íntimas
/
Claudina-2
/
Antígenos de Bactérias
Limite:
Humans
Idioma:
En
Ano de publicação:
2013
Tipo de documento:
Article