Your browser doesn't support javascript.
loading
GLUT1 deficiency in cardiomyocytes does not accelerate the transition from compensated hypertrophy to heart failure.
Pereira, Renata O; Wende, Adam R; Olsen, Curtis; Soto, Jamie; Rawlings, Tenley; Zhu, Yi; Riehle, Christian; Abel, E Dale.
Afiliação
  • Pereira RO; Division of Endocrinology, Metabolism and Diabetes, and Program in Molecular Medicine, University of Utah School of Medicine, Salt Lake City, UT, 84112, USA; Fraternal Order of Eagles Diabetes Research Center and Division of Endocrinology and Metabolism, Carver College of Medicine, University of Iow
  • Wende AR; Division of Endocrinology, Metabolism and Diabetes, and Program in Molecular Medicine, University of Utah School of Medicine, Salt Lake City, UT, 84112, USA.
  • Olsen C; Division of Endocrinology, Metabolism and Diabetes, and Program in Molecular Medicine, University of Utah School of Medicine, Salt Lake City, UT, 84112, USA.
  • Soto J; Division of Endocrinology, Metabolism and Diabetes, and Program in Molecular Medicine, University of Utah School of Medicine, Salt Lake City, UT, 84112, USA; Fraternal Order of Eagles Diabetes Research Center and Division of Endocrinology and Metabolism, Carver College of Medicine, University of Iow
  • Rawlings T; Division of Endocrinology, Metabolism and Diabetes, and Program in Molecular Medicine, University of Utah School of Medicine, Salt Lake City, UT, 84112, USA.
  • Zhu Y; Division of Endocrinology, Metabolism and Diabetes, and Program in Molecular Medicine, University of Utah School of Medicine, Salt Lake City, UT, 84112, USA.
  • Riehle C; Division of Endocrinology, Metabolism and Diabetes, and Program in Molecular Medicine, University of Utah School of Medicine, Salt Lake City, UT, 84112, USA; Fraternal Order of Eagles Diabetes Research Center and Division of Endocrinology and Metabolism, Carver College of Medicine, University of Iow
  • Abel ED; Division of Endocrinology, Metabolism and Diabetes, and Program in Molecular Medicine, University of Utah School of Medicine, Salt Lake City, UT, 84112, USA; Fraternal Order of Eagles Diabetes Research Center and Division of Endocrinology and Metabolism, Carver College of Medicine, University of Iow
J Mol Cell Cardiol ; 72: 95-103, 2014 Jul.
Article em En | MEDLINE | ID: mdl-24583251
Assuntos
Palavras-chave
Texto completo
Imprimir
XML
PubMed Links
Buscar no Google

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Hipertrofia Ventricular Esquerda / Miócitos Cardíacos / Transportador de Glucose Tipo 1 / Insuficiência Cardíaca / Mitocôndrias Cardíacas / Miocárdio Limite: Animals Idioma: En Ano de publicação: 2014 Tipo de documento: Article
Texto completo
Imprimir
XML
PubMed Links
Buscar no Google

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Hipertrofia Ventricular Esquerda / Miócitos Cardíacos / Transportador de Glucose Tipo 1 / Insuficiência Cardíaca / Mitocôndrias Cardíacas / Miocárdio Limite: Animals Idioma: En Ano de publicação: 2014 Tipo de documento: Article