Your browser doesn't support javascript.
loading
Evidence for α-synuclein prions causing multiple system atrophy in humans with parkinsonism.
Prusiner, Stanley B; Woerman, Amanda L; Mordes, Daniel A; Watts, Joel C; Rampersaud, Ryan; Berry, David B; Patel, Smita; Oehler, Abby; Lowe, Jennifer K; Kravitz, Stephanie N; Geschwind, Daniel H; Glidden, David V; Halliday, Glenda M; Middleton, Lefkos T; Gentleman, Steve M; Grinberg, Lea T; Giles, Kurt.
Afiliação
  • Prusiner SB; Institute for Neurodegenerative Diseases, University of California, San Francisco, CA 94143; Department of Neurology, University of California, San Francisco, CA 94143; Department of Biochemistry and Biophysics, University of California, San Francisco, CA 94143; stanley@ind.ucsf.edu.
  • Woerman AL; Institute for Neurodegenerative Diseases, University of California, San Francisco, CA 94143;
  • Mordes DA; C. S. Kubik Laboratory for Neuropathology, Department of Pathology, Massachusetts General Hospital, Boston, MA 02114;
  • Watts JC; Institute for Neurodegenerative Diseases, University of California, San Francisco, CA 94143; Department of Neurology, University of California, San Francisco, CA 94143;
  • Rampersaud R; Institute for Neurodegenerative Diseases, University of California, San Francisco, CA 94143;
  • Berry DB; Institute for Neurodegenerative Diseases, University of California, San Francisco, CA 94143;
  • Patel S; Institute for Neurodegenerative Diseases, University of California, San Francisco, CA 94143;
  • Oehler A; Department of Pathology, University of California, San Francisco, CA 94143;
  • Lowe JK; Center for Neurobehavioral Genetics, Center for Autism Research and Treatment, and Department of Neurology, University of California, Los Angeles, CA 90095;
  • Kravitz SN; Center for Neurobehavioral Genetics, Center for Autism Research and Treatment, and Department of Neurology, University of California, Los Angeles, CA 90095;
  • Geschwind DH; Center for Neurobehavioral Genetics, Center for Autism Research and Treatment, and Department of Neurology, University of California, Los Angeles, CA 90095; Department of Human Genetics, University of California, Los Angeles, CA 90095;
  • Glidden DV; Department of Epidemiology and Biostatistics, University of California, San Francisco, CA 94143;
  • Halliday GM; School of Medical Science, Faculty of Medicine, University of New South Wales, and Neuroscience Research Australia, Randwick, NSW 2031, Australia;
  • Middleton LT; Ageing Research Unit, School of Public Health, Imperial College London, London SW7 2AZ, United Kingdom;
  • Gentleman SM; Centre for Neuroinflammation and Neurodegeneration, Department of Medicine, Imperial College London, London SW7 2AZ, United Kingdom;
  • Grinberg LT; Department of Neurology, University of California, San Francisco, CA 94143; Memory and Aging Center, University of California, San Francisco, CA 94143.
  • Giles K; Institute for Neurodegenerative Diseases, University of California, San Francisco, CA 94143; Department of Neurology, University of California, San Francisco, CA 94143;
Proc Natl Acad Sci U S A ; 112(38): E5308-17, 2015 Sep 22.
Article em En | MEDLINE | ID: mdl-26324905
Assuntos
Palavras-chave
Texto completo
Imprimir
XML
PubMed Links
Buscar no Google

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Príons / Atrofia de Múltiplos Sistemas / Transtornos Parkinsonianos / Alfa-Sinucleína Tipo de estudo: Prognostic_studies Limite: Aged / Animals / Female / Humans / Male / Middle aged Idioma: En Ano de publicação: 2015 Tipo de documento: Article
Texto completo
Imprimir
XML
PubMed Links
Buscar no Google

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Príons / Atrofia de Múltiplos Sistemas / Transtornos Parkinsonianos / Alfa-Sinucleína Tipo de estudo: Prognostic_studies Limite: Aged / Animals / Female / Humans / Male / Middle aged Idioma: En Ano de publicação: 2015 Tipo de documento: Article