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Celecoxib induces apoptosis but up-regulates VEGF via endoplasmic reticulum stress in human colorectal cancer in vitro and in vivo.
Xu, Bingfei; Wang, Yu; Yang, Jing; Zhang, Zhengfeng; Zhang, Ying; Du, Hansong.
Afiliação
  • Xu B; Cancer Center, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, People's Republic of China.
  • Wang Y; Department of Biochemistry and Molecular Biology, School of Basic Medicine, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, People's Republic of China.
  • Yang J; Department of Infectious Disease, Renmin Hospital, Hubei University of Medicine, Shiyan, People's Republic of China.
  • Zhang Z; Department of Biochemistry and Molecular Biology, School of Basic Medicine, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, People's Republic of China.
  • Zhang Y; Department of Biochemistry and Molecular Biology, School of Basic Medicine, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, People's Republic of China.
  • Du H; Department of Gastrointestinal Surgery, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, 1277 Jiefang Avenue, Wuhan, 430030, People's Republic of China. u200910446@hust.edu.cn.
Cancer Chemother Pharmacol ; 77(4): 797-806, 2016 Apr.
Article em En | MEDLINE | ID: mdl-26931344

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Apoptose / Fator A de Crescimento do Endotélio Vascular / Inibidores de Ciclo-Oxigenase 2 / Estresse do Retículo Endoplasmático / Celecoxib Tipo de estudo: Prognostic_studies Limite: Animals / Female / Humans Idioma: En Ano de publicação: 2016 Tipo de documento: Article

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Apoptose / Fator A de Crescimento do Endotélio Vascular / Inibidores de Ciclo-Oxigenase 2 / Estresse do Retículo Endoplasmático / Celecoxib Tipo de estudo: Prognostic_studies Limite: Animals / Female / Humans Idioma: En Ano de publicação: 2016 Tipo de documento: Article