Iron depletion in HCT116 cells diminishes the upregulatory effect of phenethyl isothiocyanate on heme oxygenase-1.

Bolloskis, Michael P; Carvalho, Fabiana P; Loo, George

Bolloskis, Michael P; Carvalho, Fabiana P; Loo, George.

Afiliação

Bolloskis MP; Cellular and Molecular Nutrition Research Laboratory, Department of Nutrition, University of North Carolina at Greensboro, Greensboro, NC 27412, USA.
Carvalho FP; Cellular and Molecular Nutrition Research Laboratory, Department of Nutrition, University of North Carolina at Greensboro, Greensboro, NC 27412, USA.
Loo G; Cellular and Molecular Nutrition Research Laboratory, Department of Nutrition, University of North Carolina at Greensboro, Greensboro, NC 27412, USA. Electronic address: g_loo@uncg.edu.

Toxicol Appl Pharmacol ; 297: 22-31, 2016 Apr 15.

Article em En | MEDLINE | ID: mdl-26945724

Assuntos

Heme Oxigenase-1/metabolismo; Ferro/metabolismo; Isotiocianatos/farmacologia; Acetofenonas/farmacologia; Células HCT116; Heme Oxigenase-1/genética; Humanos; Deficiências de Ferro; NADPH Oxidases/antagonistas & inibidores; NADPH Oxidases/metabolismo; Fator 2 Relacionado a NF-E2/metabolismo; Oniocompostos/farmacologia; Regulação para Cima

Palavras-chave

Deferoxamine; Heme oxygenase-1; Iron; Phenethyl isothiocyanate

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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Isotiocianatos / Heme Oxigenase-1 / Ferro Limite: Humans Idioma: En Ano de publicação: 2016 Tipo de documento: Article

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