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CCL2 expression is mediated by type I IFN receptor and recruits NK and T cells to the lung during MVA infection.
Lehmann, Michael H; Torres-Domínguez, Lino E; Price, Philip J R; Brandmüller, Christine; Kirschning, Carsten J; Sutter, Gerd.
Afiliação
  • Lehmann MH; Institute for Infectious Diseases and Zoonoses, Ludwig-Maximilians-Universität München, Munich, Germany; Division of Virology, Paul-Ehrlich-Institut, Langen, Germany; and michael.lehmann@lmu.de.
  • Torres-Domínguez LE; Institute for Infectious Diseases and Zoonoses, Ludwig-Maximilians-Universität München, Munich, Germany;
  • Price PJ; Institute for Infectious Diseases and Zoonoses, Ludwig-Maximilians-Universität München, Munich, Germany;
  • Brandmüller C; Institute for Infectious Diseases and Zoonoses, Ludwig-Maximilians-Universität München, Munich, Germany;
  • Kirschning CJ; Institute of Medical Microbiology, University of Duisburg-Essen, Essen, Germany.
  • Sutter G; Institute for Infectious Diseases and Zoonoses, Ludwig-Maximilians-Universität München, Munich, Germany;
J Leukoc Biol ; 99(6): 1057-64, 2016 06.
Article em En | MEDLINE | ID: mdl-26992431

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Vaccinia virus / Células Matadoras Naturais / Linfócitos T / Quimiocina CCL2 / Receptor de Interferon alfa e beta / Pulmão Limite: Animals Idioma: En Ano de publicação: 2016 Tipo de documento: Article

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Vaccinia virus / Células Matadoras Naturais / Linfócitos T / Quimiocina CCL2 / Receptor de Interferon alfa e beta / Pulmão Limite: Animals Idioma: En Ano de publicação: 2016 Tipo de documento: Article