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Dysfunction of mitochondria and deformed gap junctions in the heart of IL-18-deficient mice.
Li, Wen; Jin, Denan; Hata, Masaki; Takai, Shinji; Yamanishi, Kyosuke; Shen, Weili; El-Darawish, Yosif; Yamanishi, Hiromichi; Okamura, Haruki.
Afiliação
  • Li W; Laboratory of Tumor Immunology and Cell Therapy, Hyogo College of Medicine, Hyogo, Japan;
  • Jin D; Department of Pharmacology, Osaka Medical College, Osaka, Japan;
  • Hata M; Laboratory of Tumor Immunology and Cell Therapy, Hyogo College of Medicine, Hyogo, Japan;
  • Takai S; Department of Pharmacology, Osaka Medical College, Osaka, Japan;
  • Yamanishi K; Department of Neuropsychiatry, Hyogo College of Medicine, Hyogo, Japan;
  • Shen W; Shanghai Key Laboratory of Hypertension, Shanghai, China; and.
  • El-Darawish Y; Laboratory of Tumor Immunology and Cell Therapy, Hyogo College of Medicine, Hyogo, Japan;
  • Yamanishi H; Hirakata General Hospital for Developmental Disorders, Osaka, Japan.
  • Okamura H; Laboratory of Tumor Immunology and Cell Therapy, Hyogo College of Medicine, Hyogo, Japan; haruoka@hyo-med.ac.jp.
Am J Physiol Heart Circ Physiol ; 311(2): H313-25, 2016 08 01.
Article em En | MEDLINE | ID: mdl-27288439
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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Autofagia / Junções Comunicantes / Interleucina-18 / Miócitos Cardíacos / Mitocôndrias Cardíacas Limite: Animals Idioma: En Ano de publicação: 2016 Tipo de documento: Article
Texto completo
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XML
PubMed Links
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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Autofagia / Junções Comunicantes / Interleucina-18 / Miócitos Cardíacos / Mitocôndrias Cardíacas Limite: Animals Idioma: En Ano de publicação: 2016 Tipo de documento: Article