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IL-17A-producing CD30(+) Vδ1 T cells drive inflammation-induced cancer progression.
Kimura, Yoshitaka; Nagai, Nao; Tsunekawa, Naoki; Sato-Matsushita, Marimo; Yoshimoto, Takayuki; Cua, Daniel J; Iwakura, Yoichiro; Yagita, Hideo; Okada, Futoshi; Tahara, Hideaki; Saiki, Ikuo; Irimura, Tatsuro; Hayakawa, Yoshihiro.
Afiliação
  • Kimura Y; Laboratory of Cancer Biology and Molecular Immunology, Graduate School of Pharmaceutical Sciences, University of Tokyo, Bunkyo-ku, Tokyo, Japan.
  • Nagai N; Laboratory of Cancer Biology and Molecular Immunology, Graduate School of Pharmaceutical Sciences, University of Tokyo, Bunkyo-ku, Tokyo, Japan.
  • Tsunekawa N; Laboratory of Cancer Biology and Molecular Immunology, Graduate School of Pharmaceutical Sciences, University of Tokyo, Bunkyo-ku, Tokyo, Japan.
  • Sato-Matsushita M; Department of Surgery and Bioengineering, Institute of Medical Science, University of Tokyo, Minato-ku, Tokyo, Japan.
  • Yoshimoto T; Department of Immunoregulation, Institute of Medical Science, Tokyo Medical University, Shinjuku-ku, Tokyo, Japan.
  • Cua DJ; Pathway Biology, Merck Research Laboratories, Palo Alto, California, USA.
  • Iwakura Y; Center for Animal Disease Models, Research Institute for Biomedical Sciences, Tokyo University of Science, Noda, Chiba, Japan.
  • Yagita H; Department of Immunology, Juntendo University School of Medicine, Bunkyo-ku, Tokyo, Japan.
  • Okada F; Division of Pathological Biochemistry, Tottori University Faculty of Medicine, Yonago, Tottori, Japan.
  • Tahara H; Chromosome Engineering Research Center, Tottori University, Yonago, Tottori, Japan.
  • Saiki I; Department of Surgery and Bioengineering, Institute of Medical Science, University of Tokyo, Minato-ku, Tokyo, Japan.
  • Irimura T; Division of Pathogenic Biochemistry, Department of Bioscience, Institute of Natural Medicine, University of Toyama, Toyama, Toyama, Japan.
  • Hayakawa Y; Laboratory of Cancer Biology and Molecular Immunology, Graduate School of Pharmaceutical Sciences, University of Tokyo, Bunkyo-ku, Tokyo, Japan.
Cancer Sci ; 107(9): 1206-14, 2016 Sep.
Article em En | MEDLINE | ID: mdl-27384869
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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Subpopulações de Linfócitos T / Antígeno Ki-1 / Interleucina-17 / Inflamação / Neoplasias Tipo de estudo: Prognostic_studies Limite: Animals Idioma: En Ano de publicação: 2016 Tipo de documento: Article
Texto completo
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XML
PubMed Links
Buscar no Google

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Subpopulações de Linfócitos T / Antígeno Ki-1 / Interleucina-17 / Inflamação / Neoplasias Tipo de estudo: Prognostic_studies Limite: Animals Idioma: En Ano de publicação: 2016 Tipo de documento: Article