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Inhibition of Bromodomain and Extraterminal Domain Family Proteins Ameliorates Experimental Renal Damage.
Suarez-Alvarez, Beatriz; Morgado-Pascual, José Luis; Rayego-Mateos, Sandra; Rodriguez, Ramon M; Rodrigues-Diez, Raul; Cannata-Ortiz, Pablo; Sanz, Ana B; Egido, Jesus; Tharaux, Pierre-Louis; Ortiz, Alberto; Lopez-Larrea, Carlos; Ruiz-Ortega, Marta.
Afiliação
  • Suarez-Alvarez B; Cellular Biology in Renal Diseases Laboratory, Nephrology Department and.
  • Morgado-Pascual JL; Cellular Biology in Renal Diseases Laboratory, Nephrology Department and.
  • Rayego-Mateos S; Cellular Biology in Renal Diseases Laboratory, Nephrology Department and srayego@fjd.es.
  • Rodriguez RM; Immunology Department, Hospital Universitario Central de Asturias, REDINREN, Oviedo, Spain.
  • Rodrigues-Diez R; Nephrology Department, Instituto de Investigación Sanitaria del Hospital Universitario La Paz (IdiPAZ), Madrid, REDINREN, Madrid, Spain.
  • Cannata-Ortiz P; Anatomy Department, Fundación Jiménez Díaz, Madrid, Spain.
  • Sanz AB; Dialysis Unit, Instituto de Investigación Sanitaria (IIS) Fundación Jiménez Díaz, Nephrology Department, School of Medicine, Universidad Autónoma Madrid, Renal Research Retics (REDINREN), Madrid, Spain.
  • Egido J; IIS-Fundación Jiménez Díaz, School of Medicine, Universidad Autónoma Madrid, Madrid, Spain; and.
  • Tharaux PL; Paris Cardiovascular Research Centre (PARCC), Institut National de la Santé et de la Recherche Médicale (INSERM), Paris, France.
  • Ortiz A; Dialysis Unit, Instituto de Investigación Sanitaria (IIS) Fundación Jiménez Díaz, Nephrology Department, School of Medicine, Universidad Autónoma Madrid, Renal Research Retics (REDINREN), Madrid, Spain.
  • Lopez-Larrea C; Immunology Department, Hospital Universitario Central de Asturias, REDINREN, Oviedo, Spain.
  • Ruiz-Ortega M; Cellular Biology in Renal Diseases Laboratory, Nephrology Department and.
J Am Soc Nephrol ; 28(2): 504-519, 2017 02.
Article em En | MEDLINE | ID: mdl-27436852
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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Azepinas / Fatores de Transcrição / Triazóis / Proteínas Nucleares / Proteínas Cromossômicas não Histona / Nefropatias Tipo de estudo: Etiology_studies / Prognostic_studies Limite: Animals Idioma: En Ano de publicação: 2017 Tipo de documento: Article
Texto completo
Imprimir
XML
PubMed Links
Buscar no Google

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Azepinas / Fatores de Transcrição / Triazóis / Proteínas Nucleares / Proteínas Cromossômicas não Histona / Nefropatias Tipo de estudo: Etiology_studies / Prognostic_studies Limite: Animals Idioma: En Ano de publicação: 2017 Tipo de documento: Article