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AP-1 Inhibition by SR 11302 Protects Human Hepatoma HepG2 Cells from Bile Acid-Induced Cytotoxicity by Restoring the NOS-3 Expression.
González-Rubio, Sandra; Linares, Clara I; Aguilar-Melero, Patricia; Rodríguez-Perálvarez, Manuel; Montero-Álvarez, José L; de la Mata, Manuel; Ferrín, Gustavo.
Afiliação
  • González-Rubio S; Maimonides Institute of Biomedical Research in Córdoba (IMIBIC), Reina Sofía University Hospital, University of Córdoba, Córdoba, Spain.
  • Linares CI; Maimonides Institute of Biomedical Research in Córdoba (IMIBIC), Reina Sofía University Hospital, University of Córdoba, Córdoba, Spain.
  • Aguilar-Melero P; Maimonides Institute of Biomedical Research in Córdoba (IMIBIC), Reina Sofía University Hospital, University of Córdoba, Córdoba, Spain.
  • Rodríguez-Perálvarez M; Maimonides Institute of Biomedical Research in Córdoba (IMIBIC), Reina Sofía University Hospital, University of Córdoba, Córdoba, Spain.
  • Montero-Álvarez JL; Biomedical Research Centre Network, Digestive and Liver Diseases (CIBEREHD), Instituto de Salud Carlos III, Córdoba, Spain.
  • de la Mata M; Maimonides Institute of Biomedical Research in Córdoba (IMIBIC), Reina Sofía University Hospital, University of Córdoba, Córdoba, Spain.
  • Ferrín G; Biomedical Research Centre Network, Digestive and Liver Diseases (CIBEREHD), Instituto de Salud Carlos III, Córdoba, Spain.
PLoS One ; 11(8): e0160525, 2016.
Article em En | MEDLINE | ID: mdl-27490694
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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Retinoides / Regulação para Baixo / Apoptose / Fator de Transcrição AP-1 / Óxido Nítrico Sintase Tipo III / Ácido Glicoquenodesoxicólico Tipo de estudo: Prognostic_studies Limite: Humans Idioma: En Ano de publicação: 2016 Tipo de documento: Article
Texto completo
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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Retinoides / Regulação para Baixo / Apoptose / Fator de Transcrição AP-1 / Óxido Nítrico Sintase Tipo III / Ácido Glicoquenodesoxicólico Tipo de estudo: Prognostic_studies Limite: Humans Idioma: En Ano de publicação: 2016 Tipo de documento: Article