A human-specific switch of alternatively spliced <i>AFMID</i> isoforms contributes to <i>TP53</i> mutations and tumor recurrence in hepatocellular carcinoma.

A human-specific switch of alternatively spliced AFMID isoforms contributes to TP53 mutations and tumor recurrence in hepatocellular carcinoma.

Lin, Kuan-Ting; Ma, Wai Kit; Scharner, Juergen; Liu, Yun-Ru; Krainer, Adrian R.

Afiliação

Lin KT; Cold Spring Harbor Laboratory, Cold Spring Harbor, New York 11724, USA.
Ma WK; Cold Spring Harbor Laboratory, Cold Spring Harbor, New York 11724, USA.
Scharner J; Cold Spring Harbor Laboratory, Cold Spring Harbor, New York 11724, USA.
Liu YR; Joint Biobank, Office of Human Research, Taipei Medical University, Taipei, Taiwan 11031.
Krainer AR; Cold Spring Harbor Laboratory, Cold Spring Harbor, New York 11724, USA.

Genome Res ; 2018 Feb 15.

Article em En | MEDLINE | ID: mdl-29449409

Texto completo: 1 Base de dados: MEDLINE Idioma: En Ano de publicação: 2018 Tipo de documento: Article

Texto completo: 1 Base de dados: MEDLINE Idioma: En Ano de publicação: 2018 Tipo de documento: Article