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Proteoglycan 4 regulates macrophage function without altering atherosclerotic lesion formation in a murine bone marrow-specific deletion model.
Nahon, Joya E; Hoekstra, Menno; Havik, Stefan R; Van Santbrink, Peter J; Dallinga-Thie, Geesje M; Kuivenhoven, Jan-Albert; Geerling, Janine J; Van Eck, Miranda.
Afiliação
  • Nahon JE; Division of BioTherapeutics, Leiden Academic Centre for Drug Research (LACDR), The Netherlands. Electronic address: j.e.nahon@lacdr.leidenuniv.nl.
  • Hoekstra M; Division of BioTherapeutics, Leiden Academic Centre for Drug Research (LACDR), The Netherlands.
  • Havik SR; Department of Experimental Vascular Medicine, Academic Medical Center Amsterdam, The Netherlands.
  • Van Santbrink PJ; Division of BioTherapeutics, Leiden Academic Centre for Drug Research (LACDR), The Netherlands.
  • Dallinga-Thie GM; Department of Experimental Vascular Medicine, Academic Medical Center Amsterdam, The Netherlands.
  • Kuivenhoven JA; Department of Pediatrics, University Medical Center Groningen, The Netherlands.
  • Geerling JJ; Division of BioTherapeutics, Leiden Academic Centre for Drug Research (LACDR), The Netherlands.
  • Van Eck M; Division of BioTherapeutics, Leiden Academic Centre for Drug Research (LACDR), The Netherlands.
Atherosclerosis ; 274: 120-127, 2018 07.
Article em En | MEDLINE | ID: mdl-29772480

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Proteoglicanas / Células da Medula Óssea / Artérias Carótidas / Doenças das Artérias Carótidas / Macrófagos Peritoneais / Aterosclerose / Placa Aterosclerótica / Células Espumosas Tipo de estudo: Prognostic_studies Limite: Animals / Humans / Male Idioma: En Ano de publicação: 2018 Tipo de documento: Article

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Proteoglicanas / Células da Medula Óssea / Artérias Carótidas / Doenças das Artérias Carótidas / Macrófagos Peritoneais / Aterosclerose / Placa Aterosclerótica / Células Espumosas Tipo de estudo: Prognostic_studies Limite: Animals / Humans / Male Idioma: En Ano de publicação: 2018 Tipo de documento: Article