ASH1-catalyzed H3K36 methylation drives gene repression and marks H3K27me2/3-competent chromatin.

Bicocca, Vincent T; Ormsby, Tereza; Adhvaryu, Keyur K; Honda, Shinji; Selker, Eric U

Bicocca, Vincent T; Ormsby, Tereza; Adhvaryu, Keyur K; Honda, Shinji; Selker, Eric U.

Afiliação

Bicocca VT; Institute of Molecular Biology, University of Oregon, Eugene, United States.
Ormsby T; Department of Biochemistry Faculty of Science, Charles University, Prague, Czech Republic.
Adhvaryu KK; St. George's University School of Medicine, Grenada, Caribbean.
Honda S; Faculty of Medical Sciences, University of Fukui, Fukui, Japan.
Selker EU; Institute of Molecular Biology, University of Oregon, Eugene, United States.

Elife ; 72018 11 23.

Article em En | MEDLINE | ID: mdl-30468429

Assuntos

Cromatina/metabolismo; Repressão Epigenética; Histona-Lisina N-Metiltransferase/metabolismo; Histonas/metabolismo; Neurospora crassa/enzimologia; Processamento de Proteína Pós-Traducional; Lisina/metabolismo; Metilação; Neurospora crassa/genética; Neurospora crassa/metabolismo

Palavras-chave

H3K27 methylation; H3K36 methylation; N. crassa; Set2; chromosomes; gene expression; gene silencing; genetics; genomics; heterochromatin; transcription

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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Cromatina / Histonas / Processamento de Proteína Pós-Traducional / Histona-Lisina N-Metiltransferase / Repressão Epigenética / Neurospora crassa Idioma: En Ano de publicação: 2018 Tipo de documento: Article

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