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Acetaminophen cytotoxicity in HepG2 cells is associated with a decoupling of glycolysis from the TCA cycle, loss of NADPH production, and suppression of anabolism.
Behrends, Volker; Giskeødegård, Guro F; Bravo-Santano, Natalia; Letek, Michal; Keun, Hector C.
Afiliação
  • Behrends V; Health Science Research Centre, Department of Life Sciences, University of Roehampton, London, UK. volker.behrends@roehampton.ac.uk.
  • Giskeødegård GF; Department of Circulation and Medical Imaging, Faculty of Medicine and Health Sciences, The Norwegian University of Science and Technology (NTNU), Trondheim, Norway.
  • Bravo-Santano N; Health Science Research Centre, Department of Life Sciences, University of Roehampton, London, UK.
  • Letek M; Health Science Research Centre, Department of Life Sciences, University of Roehampton, London, UK.
  • Keun HC; Department of Surgery and Cancer, Faculty of Medicine, Imperial College London, London, UK. h.keun@imperial.ac.uk.
Arch Toxicol ; 93(2): 341-353, 2019 02.
Article em En | MEDLINE | ID: mdl-30552463
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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Glicólise / Acetaminofen / Metabolismo / NADP Tipo de estudo: Risk_factors_studies Limite: Humans Idioma: En Ano de publicação: 2019 Tipo de documento: Article
Texto completo
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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Glicólise / Acetaminofen / Metabolismo / NADP Tipo de estudo: Risk_factors_studies Limite: Humans Idioma: En Ano de publicação: 2019 Tipo de documento: Article