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miR-29a attenuates cardiac hypertrophy through inhibition of PPARδ expression.
Zhang, Si; Yin, Zhongnan; Dai, Fei-Fei; Wang, Hao; Zhou, Meng-Jiao; Yang, Ming-Hui; Zhang, Shu-Feng; Fu, Zhi-Feng; Mei, Ying-Wu; Zang, Ming-Xi; Xue, Lixiang.
Afiliação
  • Zhang S; Department of Biochemistry & Molecular Biology, School of Basic Medical Sciences, Zhengzhou University, Zhengzhou City, Henan, Peoples's Republic of China.
  • Yin Z; Department of Clinical Laboratory, The Zhengzhou Central Hospital Affiliated to Zhengzhou University, Zhengzhou City, Henan, Peoples's Republic of China.
  • Dai FF; Biobank, Peking University Third Hospital, Beijing, Peoples's Republic of China.
  • Wang H; Department of Biochemistry & Molecular Biology, School of Basic Medical Sciences, Zhengzhou University, Zhengzhou City, Henan, Peoples's Republic of China.
  • Zhou MJ; Medical Research Center, Peking University Third Hospital, Beijing, Peoples's Republic of China.
  • Yang MH; Department of Biochemistry & Molecular Biology, School of Basic Medical Sciences, Zhengzhou University, Zhengzhou City, Henan, Peoples's Republic of China.
  • Zhang SF; Department of Biochemistry & Molecular Biology, School of Basic Medical Sciences, Zhengzhou University, Zhengzhou City, Henan, Peoples's Republic of China.
  • Fu ZF; Department of Pediatrics, The People's Hospital of Henan Province, Zhengzhou, Henan, Peoples's Republic of China.
  • Mei YW; Statistics and Actuarial Science Department, Faculty of Science, The University of Hong Kong, Pok Fu Lam, Hong Kong SAR, Peoples's Republic of China.
  • Zang MX; Department of Biochemistry & Molecular Biology, School of Basic Medical Sciences, Zhengzhou University, Zhengzhou City, Henan, Peoples's Republic of China.
  • Xue L; Department of Biochemistry & Molecular Biology, School of Basic Medical Sciences, Zhengzhou University, Zhengzhou City, Henan, Peoples's Republic of China.
J Cell Physiol ; 234(8): 13252-13262, 2019 08.
Article em En | MEDLINE | ID: mdl-30580435
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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Regulação da Expressão Gênica / Fator Natriurético Atrial / Cardiomegalia / Receptores Citoplasmáticos e Nucleares / MicroRNAs Tipo de estudo: Prognostic_studies / Risk_factors_studies Limite: Animals Idioma: En Ano de publicação: 2019 Tipo de documento: Article
Texto completo
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PubMed Links
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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Regulação da Expressão Gênica / Fator Natriurético Atrial / Cardiomegalia / Receptores Citoplasmáticos e Nucleares / MicroRNAs Tipo de estudo: Prognostic_studies / Risk_factors_studies Limite: Animals Idioma: En Ano de publicação: 2019 Tipo de documento: Article