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Crouzon syndrome mouse model exhibits cartilage hyperproliferation and defective segmentation in the developing trachea.
Hines, Elizabeth A; Jones, Mary-Kayt N; Harvey, Julie F; Perlyn, Chad; Ornitz, David M; Sun, Xin; Verheyden, Jamie M.
Afiliação
  • Hines EA; Laboratory of Genetics, University of Wisconsin, Madison, WI, 53706, USA.
  • Jones MN; Laboratory of Genetics, University of Wisconsin, Madison, WI, 53706, USA.
  • Harvey JF; Laboratory of Genetics, University of Wisconsin, Madison, WI, 53706, USA.
  • Perlyn C; Department of Surgery, Florida International University College of Medicine, Miami, FL, 33199, USA.
  • Ornitz DM; Department of Developmental Biology, Washington University School of Medicine, St. Louis, MO, 63110, USA.
  • Sun X; Laboratory of Genetics, University of Wisconsin, Madison, WI, 53706, USA. xinsun@ucsd.edu.
  • Verheyden JM; Department of Pediatrics, University of California-San Diego, La Jolla, CA, 92093, USA. xinsun@ucsd.edu.
Sci China Life Sci ; 62(10): 1375-1380, 2019 Oct.
Article em En | MEDLINE | ID: mdl-31463736

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Traqueia / Cartilagem / Disostose Craniofacial / Receptor Tipo 2 de Fator de Crescimento de Fibroblastos Tipo de estudo: Prognostic_studies Limite: Animals / Female / Humans / Pregnancy Idioma: En Ano de publicação: 2019 Tipo de documento: Article

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Traqueia / Cartilagem / Disostose Craniofacial / Receptor Tipo 2 de Fator de Crescimento de Fibroblastos Tipo de estudo: Prognostic_studies Limite: Animals / Female / Humans / Pregnancy Idioma: En Ano de publicação: 2019 Tipo de documento: Article