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Methionine Adenosyltransferase 1a (MAT1A) Enhances Cell Survival During Chemotherapy Treatment and is Associated with Drug Resistance in Bladder Cancer PDX Mice.
Martin, Kelly A; Hum, Nicholas R; Sebastian, Aimy; He, Wei; Siddiqui, Salma; Ghosh, Paramita M; Pan, Chong-Xian; de Vere White, Ralph; Loots, Gabriela G.
Afiliação
  • Martin KA; Lawrence Livermore National Laboratory, Physical and Life Sciences Directorate, Livermore, CA 94550, USA. martin249@llnl.gov.
  • Hum NR; Department of Biochemistry & Molecular Biology, Georgetown University, Washington, DC 20057, USA. martin249@llnl.gov.
  • Sebastian A; Lawrence Livermore National Laboratory, Physical and Life Sciences Directorate, Livermore, CA 94550, USA. hum3@llnl.gov.
  • He W; UC Merced, School of Natural Sciences, Merced, CA 95343, USA. hum3@llnl.gov.
  • Siddiqui S; Lawrence Livermore National Laboratory, Physical and Life Sciences Directorate, Livermore, CA 94550, USA. sebastian4@llnl.gov.
  • Ghosh PM; Lawrence Livermore National Laboratory, Physical and Life Sciences Directorate, Livermore, CA 94550, USA. he4@llnl.gov.
  • Pan CX; UC Davis Comprehensive Cancer Center, Sacramento, CA 95817, USA. SALMA.SIDDIQUI@va.gov.
  • de Vere White R; UC Davis Comprehensive Cancer Center, Sacramento, CA 95817, USA. paghosh@ucdavis.edu.
  • Loots GG; UC Davis Comprehensive Cancer Center, Sacramento, CA 95817, USA. cxpan@ucdavis.edu.
Int J Mol Sci ; 20(20)2019 Oct 09.
Article em En | MEDLINE | ID: mdl-31600961

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Sobrevivência Celular / Resistencia a Medicamentos Antineoplásicos / Metionina Adenosiltransferase / Antineoplásicos Tipo de estudo: Risk_factors_studies Limite: Animals / Humans Idioma: En Ano de publicação: 2019 Tipo de documento: Article

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Sobrevivência Celular / Resistencia a Medicamentos Antineoplásicos / Metionina Adenosiltransferase / Antineoplásicos Tipo de estudo: Risk_factors_studies Limite: Animals / Humans Idioma: En Ano de publicação: 2019 Tipo de documento: Article