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Prioritizing disease and trait causal variants at the TNFAIP3 locus using functional and genomic features.
Ray, John P; de Boer, Carl G; Fulco, Charles P; Lareau, Caleb A; Kanai, Masahiro; Ulirsch, Jacob C; Tewhey, Ryan; Ludwig, Leif S; Reilly, Steven K; Bergman, Drew T; Engreitz, Jesse M; Issner, Robbyn; Finucane, Hilary K; Lander, Eric S; Regev, Aviv; Hacohen, Nir.
Afiliação
  • Ray JP; Broad Institute of MIT and Harvard, Cambridge, MA, 02142, USA.
  • de Boer CG; Broad Institute of MIT and Harvard, Cambridge, MA, 02142, USA.
  • Fulco CP; Klarman Cell Observatory, Broad Institute of MIT and Harvard, Cambridge, MA, 02142, USA.
  • Lareau CA; Broad Institute of MIT and Harvard, Cambridge, MA, 02142, USA.
  • Kanai M; Department of Systems Biology, Harvard Medical School, Boston, MA, 02115, USA.
  • Ulirsch JC; Broad Institute of MIT and Harvard, Cambridge, MA, 02142, USA.
  • Tewhey R; Program in Biological and Biomedical Sciences, Harvard Medical School, Boston, MA, 02115, USA.
  • Ludwig LS; Broad Institute of MIT and Harvard, Cambridge, MA, 02142, USA.
  • Reilly SK; Analytic and Translational Genetics Unit, Massachusetts General Hospital, Boston, MA, 02114, USA.
  • Bergman DT; Program in Bioinformatics and Integrative Genomics, Harvard Medical School, Boston, MA, 02115, USA.
  • Engreitz JM; Broad Institute of MIT and Harvard, Cambridge, MA, 02142, USA.
  • Issner R; Program in Biological and Biomedical Sciences, Harvard Medical School, Boston, MA, 02115, USA.
  • Finucane HK; Broad Institute of MIT and Harvard, Cambridge, MA, 02142, USA.
  • Lander ES; Department of Organismic and Evolutionary Biology, Harvard University, Cambridge, MA, 02138, USA.
  • Regev A; Broad Institute of MIT and Harvard, Cambridge, MA, 02142, USA.
  • Hacohen N; Broad Institute of MIT and Harvard, Cambridge, MA, 02142, USA.
Nat Commun ; 11(1): 1237, 2020 03 06.
Article em En | MEDLINE | ID: mdl-32144282
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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Doenças Autoimunes / Herança Multifatorial / Estudo de Associação Genômica Ampla / Loci Gênicos / Proteína 3 Induzida por Fator de Necrose Tumoral alfa Tipo de estudo: Prognostic_studies Limite: Humans Idioma: En Ano de publicação: 2020 Tipo de documento: Article
Texto completo
Imprimir
XML
PubMed Links
Buscar no Google

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Doenças Autoimunes / Herança Multifatorial / Estudo de Associação Genômica Ampla / Loci Gênicos / Proteína 3 Induzida por Fator de Necrose Tumoral alfa Tipo de estudo: Prognostic_studies Limite: Humans Idioma: En Ano de publicação: 2020 Tipo de documento: Article