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The SQSTM1-NUP214 fusion protein interacts with Crm1, activates Hoxa and Meis1 genes, and drives leukemogenesis in mice.
Lavau, Catherine P; Aumann, Waitman K; Sze, Sei-Gyung K; Gupta, Veerain; Ripple, Katelyn; Port, Sarah A; Kehlenbach, Ralph H; Wechsler, Daniel S.
Afiliação
  • Lavau CP; Department of Neurosurgery, Duke University Medical Center, Durham, North Carolina, United States of America.
  • Aumann WK; Aflac Cancer & Blood Disorders Center, Children's Healthcare of Atlanta, Atlanta, Georgia, United States of America.
  • Sze SK; Department of Pediatrics, Emory University School of Medicine, Atlanta, Georgia, United States of America.
  • Gupta V; Maine Children's Cancer Program, Scarborough, Maine, United States of America.
  • Ripple K; Department of Pediatrics, Duke University Medical Center, Durham, North Carolina, United States of America.
  • Port SA; Department of Pediatrics, Duke University Medical Center, Durham, North Carolina, United States of America.
  • Kehlenbach RH; Department of Molecular Biology, Princeton University, Princeton, New Jersey, United States of America.
  • Wechsler DS; Department of Molecular Biology, Faculty of Medicine and the Göttingen Center for Molecular Biosciences (GZMB), Göttingen, Germany.
PLoS One ; 15(4): e0232036, 2020.
Article em En | MEDLINE | ID: mdl-32343715
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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Leucemia / Proteínas de Fusão Oncogênica / Receptores Citoplasmáticos e Nucleares / Proteínas de Homeodomínio / Carioferinas / Complexo de Proteínas Formadoras de Poros Nucleares / Proteína Sequestossoma-1 / Proteína Meis1 Limite: Animals Idioma: En Ano de publicação: 2020 Tipo de documento: Article
Texto completo
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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Leucemia / Proteínas de Fusão Oncogênica / Receptores Citoplasmáticos e Nucleares / Proteínas de Homeodomínio / Carioferinas / Complexo de Proteínas Formadoras de Poros Nucleares / Proteína Sequestossoma-1 / Proteína Meis1 Limite: Animals Idioma: En Ano de publicação: 2020 Tipo de documento: Article