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Murine iNKT cells are depleted by liver damage via activation of P2RX7.
Bovens, Astrid A; Wesselink, Thomas H; Behr, Felix M; Kragten, Natasja A M; van Lier, René A W; van Gisbergen, Klaas P J M; Stark, Regina.
Afiliação
  • Bovens AA; Department of Hematopoiesis, Sanquin Research and Landsteiner Laboratory, Amsterdam UMC, University of Amsterdam, Amsterdam, The Netherlands.
  • Wesselink TH; Department of Hematopoiesis, Sanquin Research and Landsteiner Laboratory, Amsterdam UMC, University of Amsterdam, Amsterdam, The Netherlands.
  • Behr FM; Department of Hematopoiesis, Sanquin Research and Landsteiner Laboratory, Amsterdam UMC, University of Amsterdam, Amsterdam, The Netherlands.
  • Kragten NAM; Department of Experimental Immunology, Amsterdam UMC, University of Amsterdam, Amsterdam, The Netherlands.
  • van Lier RAW; Department of Hematopoiesis, Sanquin Research and Landsteiner Laboratory, Amsterdam UMC, University of Amsterdam, Amsterdam, The Netherlands.
  • van Gisbergen KPJM; Department of Hematopoiesis, Sanquin Research and Landsteiner Laboratory, Amsterdam UMC, University of Amsterdam, Amsterdam, The Netherlands.
  • Stark R; Department of Hematopoiesis, Sanquin Research and Landsteiner Laboratory, Amsterdam UMC, University of Amsterdam, Amsterdam, The Netherlands.
Eur J Immunol ; 50(10): 1515-1524, 2020 10.
Article em En | MEDLINE | ID: mdl-32390174
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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Células T Matadoras Naturais / Doença Hepática Induzida por Substâncias e Drogas / Receptores Purinérgicos P2X7 / Fígado / Acetaminofen Tipo de estudo: Prognostic_studies Limite: Animals / Humans Idioma: En Ano de publicação: 2020 Tipo de documento: Article
Texto completo
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PubMed Links
Buscar no Google

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Células T Matadoras Naturais / Doença Hepática Induzida por Substâncias e Drogas / Receptores Purinérgicos P2X7 / Fígado / Acetaminofen Tipo de estudo: Prognostic_studies Limite: Animals / Humans Idioma: En Ano de publicação: 2020 Tipo de documento: Article