Disturbed intramitochondrial phosphatidic acid transport impairs cellular stress signaling.

Eiyama, Akinori; Aaltonen, Mari J; Nolte, Hendrik; Tatsuta, Takashi; Langer, Thomas

Eiyama, Akinori; Aaltonen, Mari J; Nolte, Hendrik; Tatsuta, Takashi; Langer, Thomas.

Afiliação

Eiyama A; Max-Planck-Institute for Biology of Ageing, Cologne, Germany.
Aaltonen MJ; Cologne Excellence Cluster on Cellular Stress Responses in Aging-Associated Diseases (CECAD), University of Cologne, Cologne, Germany.
Nolte H; Max-Planck-Institute for Biology of Ageing, Cologne, Germany.
Tatsuta T; Max-Planck-Institute for Biology of Ageing, Cologne, Germany.
Langer T; Max-Planck-Institute for Biology of Ageing, Cologne, Germany; Cologne Excellence Cluster on Cellular Stress Responses in Aging-Associated Diseases (CECAD), University of Cologne, Cologne, Germany. Electronic address: TLanger@age.mpg.de.

J Biol Chem ; 296: 100335, 2021.

Article em En | MEDLINE | ID: mdl-33497623

Assuntos

Mitocôndrias/metabolismo; Ácidos Fosfatídicos/metabolismo; Saccharomyces cerevisiae/metabolismo; Transdução de Sinais; Transporte Biológico; Alvo Mecanístico do Complexo 1 de Rapamicina/metabolismo; Proteínas de Saccharomyces cerevisiae/metabolismo; Resposta a Proteínas não Dobradas

Palavras-chave

PRELID1; TORC1; Ups1; endoplasmic reticulum; lipid transfer; mitochondria; phospholipid; unfolded protein response; yeast

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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Ácidos Fosfatídicos / Saccharomyces cerevisiae / Transdução de Sinais / Mitocôndrias Idioma: En Ano de publicação: 2021 Tipo de documento: Article

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